Brooke A. Acton, Ph.D.
Affiliations: | Cell and Systems Biology | University of Toronto, Toronto, ON, Canada |
Area:
Inhibitory Synaptic PlasticityGoogle:
"Brooke Acton"Mean distance: 15.67 (cluster 6) | S | N | B | C | P |
Parents
Sign in to add mentor| Melanie A. Woodin | grad student | 2013 | University of Toronto | |
| (Mechanisms of Inhibitory Synaptic Plasticity: the Regulation of KCC2.) | ||||
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Publications
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| Mahadevan V, Pressey JC, Acton BA, et al. (2014) Kainate receptors coexist in a functional complex with KCC2 and regulate chloride homeostasis in hippocampal neurons. Cell Reports. 7: 1762-70 |
| Ivakine EA, Acton BA, Mahadevan V, et al. (2013) Neto2 is a KCC2 interacting protein required for neuronal Cl- regulation in hippocampal neurons. Proceedings of the National Academy of Sciences of the United States of America. 110: 3561-6 |
| Acton BA, Mahadevan V, Mercado A, et al. (2012) Hyperpolarizing GABAergic transmission requires the KCC2 C-terminal ISO domain. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 32: 8746-51 |
| Balena T, Acton BA, Woodin MA. (2011) Activity-dependent inhibitory synaptic plasticity mediated by chloride regulation Inhibitory Synaptic Plasticity. 137-148 |
| Balena T, Acton BA, Woodin MA. (2010) GABAergic synaptic transmission regulates calcium influx during spike-timing dependent plasticity. Frontiers in Synaptic Neuroscience. 2: 16 |
| Balena T, Acton BA, Koval D, et al. (2008) Extracellular potassium regulates the chloride reversal potential in cultured hippocampal neurons. Brain Research. 1205: 12-20 |