Rafael Gonzalez, Ph.D.
Affiliations: | University of California, Irvine, Irvine, CA |
Area:
spinal cord injuryGoogle:
"Rafael Gonzalez"Mean distance: 17811 (cluster 11)
Parents
Sign in to add mentor| Hans Keirstead | grad student | 2006 | UC Irvine | |
| (CXCL10 plays a critical role in secondary degeneration following spinal cord injury.) | ||||
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Publications
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| Geffner LF, Santacruz P, Izurieta M, et al. (2008) Administration of autologous bone marrow stem cells into spinal cord injury patients via multiple routes is safe and improves their quality of life: Comprehensive case studies Cell Transplantation. 17: 1277-1293 |
| Gonzalez R, Hickey MJ, Espinosa JM, et al. (2007) Therapeutic neutralization of CXCL10 decreases secondary degeneration and functional deficit after spinal cord injury in mice. Regenerative Medicine. 2: 771-83 |
| Glaser J, Gonzalez R, Sadr E, et al. (2006) Neutralization of the chemokine CXCL10 reduces apoptosis and increases axon sprouting after spinal cord injury. Journal of Neuroscience Research. 84: 724-34 |
| Hardison JL, Nistor G, Gonzalez R, et al. (2006) Transplantation of glial-committed progenitor cells into a viral model of multiple sclerosis induces remyelination in the absence of an attenuated inflammatory response. Experimental Neurology. 197: 420-9 |
| Gonzalez R, Hardison JL, Glaser J, et al. (2006) The chemokine CXCL10 as a therapeutic target in animal models of neuroinflammatory disease Letters in Drug Design and Discovery. 3: 683-688 |
| Glaser J, Gonzalez R, Perreau VM, et al. (2004) Neutralization of the chemokine CXCL10 enhances tissue sparing and angiogenesis following spinal cord injury. Journal of Neuroscience Research. 77: 701-8 |
| Gonzalez R, Glaser J, Liu MT, et al. (2003) Reducing inflammation decreases secondary degeneration and functional deficit after spinal cord injury. Experimental Neurology. 184: 456-63 |