Milos Cekic, Ph.D.

Institution:
Emory University, Atlanta, GA
Area:
recovery from brain injury, progesterone, trauma, CNS plasticity
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"Milos Cekic"
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Donald G. Stein grad student 2011 Emory
 (Progesterone, Vitamin D, and the Acute Inflammatory Response After Traumatic Brain Injury in the Aged Rat.)
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Publications

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Cekic M, Johnson SJ, Bhatt VH, et al. (2012) Progesterone treatment alters neurotrophin/proneurotrophin balance and receptor expression in rats with traumatic brain injury. Restorative Neurology and Neuroscience. 30: 115-26
Stein DG, Cekic MM. (2011) Progesterone and vitamin d hormone as a biologic treatment of traumatic brain injury in the aged. Pm & R : the Journal of Injury, Function, and Rehabilitation. 3: S100-10
Cekic M, Cutler SM, VanLandingham JW, et al. (2011) Vitamin D deficiency reduces the benefits of progesterone treatment after brain injury in aged rats. Neurobiology of Aging. 32: 864-74
Cekic M, Stein DG. (2010) Traumatic brain injury and aging: is a combination of progesterone and vitamin D hormone a simple solution to a complex problem? Neurotherapeutics : the Journal of the American Society For Experimental Neurotherapeutics. 7: 81-90
Cekic M, Stein DG. (2010) Progesterone treatment for brain injury: An update Future Neurology. 5: 37-46
Cekic M, Sayeed I, Stein DG. (2009) Combination treatment with progesterone and vitamin D hormone may be more effective than monotherapy for nervous system injury and disease. Frontiers in Neuroendocrinology. 30: 158-72
VanLandingham JW, Cekic M, Cutler SM, et al. (2008) Progesterone and its metabolite allopregnanolone differentially regulate hemostatic proteins after traumatic brain injury. Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism. 28: 1786-94
Cutler SM, Cekic M, Miller DM, et al. (2007) Progesterone improves acute recovery after traumatic brain injury in the aged rat. Journal of Neurotrauma. 24: 1475-86
VanLandingham JW, Cekic M, Cutler S, et al. (2007) Neurosteroids reduce inflammation after TBI through CD55 induction. Neuroscience Letters. 425: 94-8
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