Joshua D. Ohrtman, Ph.D.
Affiliations: | University of Colorado Anschutz Medical Campus, Denver, Aurora, CO |
Area:
Excitation-contraction couplingGoogle:
"Joshua Ohrtman"Mean distance: 14.54 (cluster 46) | S | N | B | C | P |
Parents
Sign in to add mentor| Kurt G. Beam | grad student | 2010 | University of Colorado, Denver | |
| (Short-term regulatory mechanisms of calcium channels in muscle.) | ||||
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Publications
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| Polster A, Perni S, Filipova D, et al. (2017) Junctional trafficking and restoration of retrograde signaling by the cytoplasmic RyR1 domain. The Journal of General Physiology |
| Ohrtman JD, Romberg CF, Moua O, et al. (2015) Apparent lack of physical or functional interaction between CaV1.1 and its distal C terminus. The Journal of General Physiology. 145: 303-14 |
| Polster A, Ohrtman JD, Beam KG, et al. (2013) The Cytoplasmic Foot of RyR1 without the Membrane Spanning Domain Targets Junctionally and Retrogradely Enhances DHPR L-Type Ca2+ Currents Biophysical Journal. 104: 445a |
| Polster A, Ohrtman JD, Beam KG, et al. (2012) Fluorescence resonance energy transfer (FRET) indicates that association with the type I ryanodine receptor (RyR1) causes reorientation of multiple cytoplasmic domains of the dihydropyridine receptor (DHPR) α(1S) subunit. The Journal of Biological Chemistry. 287: 41560-8 |
| Polster A, Ohrtman J, Beam KG, et al. (2012) FRET Reveals Substantial Reorientation of the Cytoplasmic Interface of the Skeletal Muscle DHPR in the Presence of RyR1 Biophysical Journal. 102: 362a |
| Ohrtman JD, Beam KG, Allen PD. (2010) Both Voltage- and Ca2+-Dependent Inactivation of Cav1.2 Ca2+ Channels are Suppresed in Myotubes, Likely Because of Triad Junction Proteins other than RyR1 Biophysical Journal. 98: 516a |
| Bannister RA, Ohrtman JD. (2009) Reassessing the molecular mechanism of beta-adrenergic stimulation of cardiac L-type Ca2+ current. Channels (Austin, Tex.). 3: 146-8 |
| Ohrtman JD, Beam KG. (2009) Depolarization-induced Potentiation Of Cav1.1 Does Not Require The Distal C-terminus Biophysical Journal. 96: 184a-185a |
| Bannister RA, Ohrtman JD. (2009) Reassessing the molecular mechanism of β-adrenergic stimulation of cardiac L-type Ca2+ current Channels. 3 |
| Ohrtman J, Ritter B, Polster A, et al. (2008) Sequence differences in the IQ motifs of CaV1.1 and CaV1.2 strongly impact calmodulin binding and calcium-dependent inactivation. The Journal of Biological Chemistry. 283: 29301-11 |