April M. Weissmiller, Ph.D.
Affiliations: | Neurosciences | University of California, San Diego, La Jolla, CA |
Area:
neurodegneration, Down syndromeGoogle:
"April Weissmiller"Mean distance: 17.1 (cluster 40)
Parents
Sign in to add mentor| William C. Mobley | grad student | 2013 | UCSD | |
| (Evidence that inhibition of Gamma-secretase impairs axonal and dendritic structure and function: A necessary role for APP.) | ||||
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Publications
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| Xu W, Weissmiller AM, White JA, et al. (2016) Amyloid precursor protein-mediated endocytic pathway disruption induces axonal dysfunction and neurodegeneration. The Journal of Clinical Investigation |
| Le MH, Weissmiller AM, Monte L, et al. (2016) Functional Impact of Corticotropin-Releasing Factor Exposure on Tau Phosphorylation and Axon Transport. Plos One. 11: e0147250 |
| Selfridge A, Hyun N, Chiang CC, et al. (2015) Rat embryonic hippocampus and induced pluripotent stem cell derived cultured neurons recover from laser-induced subaxotomy. Neurophotonics. 2: 015006 |
| Nosheny RL, Belichenko PV, Busse BL, et al. (2015) Increased cortical synaptic activation of TrkB and downstream signaling markers in a mouse model of Down Syndrome. Neurobiology of Disease. 77: 173-90 |
| Weissmiller AM, Natera-Naranjo O, Reyna SM, et al. (2015) A γ-secretase inhibitor, but not a γ-secretase modulator, induced defects in BDNF axonal trafficking and signaling: evidence for a role for APP. Plos One. 10: e0118379 |
| Zhao X, Zhou Y, Weissmiller AM, et al. (2014) Real-time imaging of axonal transport of quantum dot-labeled BDNF in primary neurons. Journal of Visualized Experiments : Jove. 51899 |
| Rodrigues EM, Weissmiller AM, Goldstein LS. (2012) Enhanced β-secretase processing alters APP axonal transport and leads to axonal defects. Human Molecular Genetics. 21: 4587-601 |