Brea J. Midthune, Ph.D.
Affiliations: | Molecular Pathology | University of California, San Diego, La Jolla, CA |
Area:
http://www.researchprofiles.collexis.com/jad/expert.asp?u_id=62Google:
"Brea Midthune"Mean distance: 19.43 (cluster 31) | S | N | B | C | P |
Parents
Sign in to add mentor| Edward H. Koo | grad student | 2012 | UCSD | |
| (The Role of Amyloid Precursor Protein in Amyloid-Beta-Mediated Synaptic Dysfunction.) | ||||
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Publications
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| Midthune B, Tyan SH, Walsh JJ, et al. (2012) Deletion of the amyloid precursor-like protein 2 (APLP2) does not affect hippocampal neuron morphology or function. Molecular and Cellular Neurosciences. 49: 448-55 |
| Midthune B, Malinow R, Koo E. (2011) P3-226: Elimination of APP caspase cleavage site D664 attenuates Aβ-mediated synaptic depression Alzheimer's & Dementia. 7: S588-S588 |
| Walsh JJ, Tyan S, Midthune B, et al. (2010) P1-135: Comparison of neuronal morphology in pyramidal neurons of the CA1, dentate gyrus, and prefrontal cortex of APP knockout and APLP2 knockout mice Alzheimer's & Dementia. 6: S213-S213 |
| Tyan S, Midthune B, Eggert S, et al. (2010) P2-328: Role of amyloid precursor proteins and beta-amyloid-induced synaptic dysfunction Alzheimer's & Dementia. 6: S409-S409 |
| Eggert S, Midthune B, Cottrell B, et al. (2009) Induced dimerization of the amyloid precursor protein leads to decreased amyloid-beta protein production. The Journal of Biological Chemistry. 284: 28943-52 |