Hagit Eldar-Finkelman
Affiliations: | Tel Aviv University, Tel Aviv-Yafo, Tel Aviv District, Israel |
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"Hagit Eldar-Finkelman"Mean distance: 16.88 (cluster 6) | S | N | B | C | P |
Parents
Sign in to add mentor| Etta Livneh | grad student | 1987-1993 | Weizmann Institute (Cell Biology Tree) |
| Edwin G. Krebs | post-doc | 1993-1998 | University of Washington |
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Publications
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| Arciniegas Ruiz S, Rippin I, Eldar-Finkelman H. (2022) Prospects in GSK-3 Signaling: From Cellular Regulation to Disease Therapy. Cells. 11 |
| Avrahami L, Paz R, Dominko K, et al. (2020) GSK-3-TSC axis governs lysosomal acidification through autophagy and endocytic pathways. Cellular Signalling. 109597 |
| Eldar-Finkelman H, VanHook AM. (2016) Science Signaling Podcast for 15 November 2016: A new type of kinase inhibitor. Science Signaling. 9: pc22 |
| Grieco SF, Velmeshev D, Magistri M, et al. (2016) Ketamine up-regulates a cluster of intronic miRNAs within the serotonin receptor 2C gene by inhibiting glycogen synthase kinase-3. The World Journal of Biological Psychiatry : the Official Journal of the World Federation of Societies of Biological Psychiatry. 1-12 |
| Azoulay-Alfaguter I, Elya R, Avrahami L, et al. (2014) Combined regulation of mTORC1 and lysosomal acidification by GSK-3 suppresses autophagy and contributes to cancer cell growth. Oncogene |
| Licht-Murava A, Eldar-Finkelman H. (2012) Exploiting substrate recognition for selective inhibition of protein kinases. Current Pharmaceutical Design. 18: 2914-20 |
| Lo Monte F, Kramer T, Gu J, et al. (2012) Identification of glycogen synthase kinase-3 inhibitors with a selective sting for glycogen synthase kinase-3α. Journal of Medicinal Chemistry. 55: 4407-24 |
| Alon LT, Pietrokovski S, Barkan S, et al. (2011) Selective loss of glycogen synthase kinase-3α in birds reveals distinct roles for GSK-3 isozymes in tau phosphorylation. Febs Letters. 585: 1158-62 |
| Karyo R, Eskira Y, Pinhasov A, et al. (2010) Identification of eukaryotic elongation factor-2 as a novel cellular target of lithium and glycogen synthase kinase-3. Molecular and Cellular Neurosciences. 45: 449-55 |
| Leng S, Zhang W, Zheng Y, et al. (2010) Glycogen synthase kinase 3 beta mediates high glucose-induced ubiquitination and proteasome degradation of insulin receptor substrate 1. The Journal of Endocrinology. 206: 171-81 |