Theresa J. Barberi, Ph.D.
Affiliations: | 2011 | Molecular Cancer Biology | Duke University, Durham, NC |
Area:
cell signaling cascades that regulate cell proliferation, differentiation or functionGoogle:
"Theresa Barberi"Mean distance: 19.23 (cluster 11) | S | N | B | C | P |
Parents
Sign in to add mentorAnthony R. Means | grad student | 2011 | Duke | |
(Roles for Pin1 in Modulating Cells of the Innate Immune System.) |
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Publications
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Cui C, Barberi T, Suresh R, et al. (2021) Adoptive transfer of immature myeloid cells lacking NF-κB p50 (p50-IMC) impedes the growth of MHC-matched high-risk neuroblastoma. Molecular Oncology |
Suresh R, Barakat DJ, Barberi T, et al. (2020) NF-κB p50-deficient immature myeloid cell (p50-IMC) adoptive transfer slows the growth of murine prostate and pancreatic ductal carcinoma. Journal For Immunotherapy of Cancer. 8 |
Suresh RR, Barakat DJ, Barberi T, et al. (2020) Abstract B68: NF-kB p50-deficient immature myeloid cell (p50-IMC) adoptive transfer slows the growth of murine prostate and pancreatic ductal carcinoma Cancer Immunology Research. 8 |
Cui C, Barberi T, Suresh R, et al. (2020) 92 Adoptive transfer of immature myeloid cells lacking NF-κB p50 (p50-IMC) slows high-risk neuroblastoma tumor growth Journal For Immunotherapy of Cancer. 8: A103-A103 |
Guo H, Barberi T, Suresh R, et al. (2018) Progression from the Common Lymphoid Progenitor to B/Myeloid PreproB and ProB Precursors during B Lymphopoiesis Requires C/EBPα. Journal of Immunology (Baltimore, Md. : 1950) |
Barberi T, Martin A, Suresh R, et al. (2018) Absence of host NF-κB p50 induces murine glioblastoma tumor regression, increases survival, and decreases T-cell induction of tumor-associated macrophage M2 polarization. Cancer Immunology, Immunotherapy : Cii |
Barakat DJ, Suresh R, Barberi T, et al. (2018) Absence of myeloid Klf4 reduces prostate cancer growth with pro-atherosclerotic activation of tumor myeloid cells and infiltration of CD8 T cells. Plos One. 13: e0191188 |
Rutella S, Rumi C, Lucia MB, et al. (1999) Induction of CD69 antigen on normal CD4+ and CD8+ lymphocyte subsets and its relationship with the phenotype of responding T-cells. Cytometry. 38: 95-101 |