Phillip G. Nelson
Affiliations: | National Institutes of Health, Bethesda, MD |
Website:
http://eclipse.nichd.nih.gov/nichd/annualreport/2003/laboratory_of_developmental_neurobiology/son.htmGoogle:
"Phillip Nelson"Mean distance: 13.01 (cluster 11)
Children
Sign in to add traineeEarl Godfrey | grad student | NIH | |
Robert E. Burke | post-doc | NIH | |
Richard Douglas Fields | post-doc | NIH | |
Gerald Fischbach | post-doc | NIH | |
Bruce R. Ransom | post-doc | NIH | |
Peter Sonderegger | post-doc | Universität Zürich | |
Gary L. Westbrook | post-doc | NIH | |
Mark L. Mayer | post-doc | 1981-1986 | NIH |
Ian D. Forsythe | post-doc | 1985-1988 | NIH |
Solomon David Erulkar | research scientist | NIH | |
Ted Evans | research scientist | NIH | |
Harold Gainer | research scientist | NIH | |
Peter Latham | research scientist | NIH | |
Robert L. Macdonald | research scientist | NIH |
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Publications
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Fischbach GD, Nelson PG. (2011) Cell Culture in Neurobiology Comprehensive Physiology. 719-774 |
Lanuza MA, Besalduch N, González C, et al. (2010) Decreased phosphorylation of δ and ε subunits of the acetylcholine receptor coincides with delayed postsynaptic maturation in PKC θ deficient mouse. Experimental Neurology. 225: 183-95 |
Jia M, Li MX, Fields RD, et al. (2007) Extracellular ATP in activity-dependent remodeling of the neuromuscular junction. Developmental Neurobiology. 67: 924-32 |
Lanuza MA, Gizaw R, Viloria A, et al. (2006) Phosphorylation of the nicotinic acetylcholine receptor in myotube-cholinergic neuron cocultures. Journal of Neuroscience Research. 83: 1407-14 |
Nelson PG. (2005) Activity-dependent synapse modulation and the pathogenesis of Alzheimer disease. Current Alzheimer Research. 2: 497-506 |
Shimazu K, Takeda K, Yu ZX, et al. (2005) Multiple acute effects on the membrane potential of PC12 cells produced by nerve growth factor (NGF). Journal of Cellular Physiology. 203: 501-9 |
Yang LX, Nelson PG. (2004) Glia cell line-derived neurotrophic factor regulates the distribution of acetylcholine receptors in mouse primary skeletal muscle cells. Neuroscience. 128: 497-509 |
Li MX, Jia M, Yang LX, et al. (2004) The role of the theta isoform of protein kinase C (PKC) in activity-dependent synapse elimination: evidence from the PKC theta knock-out mouse in vivo and in vitro. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 24: 3762-9 |
Nelson PG, Lanuza MA, Jia M, et al. (2003) Phosphorylation reactions in activity-dependent synapse modification at the neuromuscular junction during development. Journal of Neurocytology. 32: 803-16 |
Lanuza MA, Garcia N, González CM, et al. (2003) Role and expression of thrombin receptor PAR-1 in muscle cells and neuromuscular junctions during the synapse elimination period in the neonatal rat. Journal of Neuroscience Research. 73: 10-21 |