Leo Pallanck

Affiliations: 
University of Washington, Seattle, Seattle, WA 
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"Leo Pallanck"
Mean distance: 15.35 (cluster 11)
 
SNBCP
Cross-listing: FlyTree

Children

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Jessica A. Golby grad student 2003 University of Washington (FlyTree)
Michael C. Babcock grad student 2004 University of Washington (FlyTree)
T F. Satterfield grad student 2005 University of Washington (FlyTree)
Megan L. Fluegel grad student 2006 University of Washington (FlyTree)
Stephen P. Voght grad student 2007 University of Washington (FlyTree)
Angela C. Poole grad student 2010 University of Washington (FlyTree)
Evelyn S. Vincow grad student 2013 University of Washington (FlyTree)
Alex Whitworth post-doc (FlyTree)
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Publications

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Sreejith P, Lolo S, Patten KR, et al. (2024) Nazo, the Drosophila homolog of the NBIA-mutated protein-c19orf12, is required for triglyceride homeostasis. Plos Genetics. 20: e1011137
Vincow ES, Thomas RE, Milstein G, et al. (2023) Glucocerebrosidase deficiency leads to neuropathology via cellular immune activation. Biorxiv : the Preprint Server For Biology
Jewett KA, Thomas RE, Phan CQ, et al. (2021) Glucocerebrosidase reduces the spread of protein aggregation in a Drosophila melanogaster model of neurodegeneration by regulating proteins trafficked by extracellular vesicles. Plos Genetics. 17: e1008859
Vincow ES, Thomas RE, Merrihew GE, et al. (2021) Slowed protein turnover in aging Drosophila reflects a shift in cellular priorities. The Journals of Gerontology. Series a, Biological Sciences and Medical Sciences
Pareek G, Pallanck LJ. (2020) Inactivation of the mitochondrial protease Afg3l2 results in severely diminished respiratory chain activity and widespread defects in mitochondrial gene expression. Plos Genetics. 16: e1009118
Andreazza S, Samstag CL, Sanchez-Martinez A, et al. (2019) Mitochondrially-targeted APOBEC1 is a potent mtDNA mutator affecting mitochondrial function and organismal fitness in Drosophila. Nature Communications. 10: 3280
Vincow ES, Thomas RE, Merrihew G, et al. (2019) Autophagy accounts for approximately one-third of mitochondrial protein turnover and is protein selective. Autophagy
Pareek G, Pallanck LJ. (2018) Inactivation of Lon protease reveals a link between mitochondrial unfolded protein stress and mitochondrial translation inhibition. Cell Death & Disease. 9: 1168
Pareek G, Thomas RE, Vincow ES, et al. (2018) Lon protease inactivation in causes unfolded protein stress and inhibition of mitochondrial translation. Cell Death Discovery. 5: 51
Thomas RE, Vincow ES, Merrihew GE, et al. (2018) Glucocerebrosidase deficiency promotes protein aggregation through dysregulation of extracellular vesicles. Plos Genetics. 14: e1007694
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