Christian W. Kreipke, Ph.D.

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2004 Wayne State University, Detroit, MI, United States 
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"Christian Kreipke"
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Paul D. Walker grad student 2004 Wayne State
 (NMDA and D1 receptor mechanisms of movement control: Differential regulatory mechanisms in the intact and dopamine depleted striatum.)
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Publications

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Maegele M, Braun M, Wafaisade A, et al. (2015) Long-term effects of enriched environment on neurofunctional outcome and CNS lesion volume after traumatic brain injury in rats Physiological Research. 64: 129-145
Wang T, Chou DY, Ding JY, et al. (2013) Reduction of brain edema and expression of aquaporins with acute ethanol treatment after traumatic brain injury. Journal of Neurosurgery. 118: 390-6
Kreipke CW, Kropinski A, Graves J, et al. (2013) New frontiers in clinical trials aimed at improving outcome following traumatic brain injury Cerebral Blood Flow, Metabolism, and Head Trauma: the Pathotrajectory of Traumatic Brain Injury. 155-163
Kropinski A, Dore-Duffy P, Kreipke CW. (2013) Situating the endothelin system in the pathotrajectory of TBI-induced changes in hemodynamics Cerebral Blood Flow, Metabolism, and Head Trauma: the Pathotrajectory of Traumatic Brain Injury. 95-133
Kane MJ, Pérez MA, Briggs DI, et al. (2013) Modeling of traumatic brain injury and its implications in studying the pathology of repeated mild impacts to the head Cerebral Blood Flow, Metabolism, and Head Trauma: the Pathotrajectory of Traumatic Brain Injury. 53-73
Betrus C, Kreipke CW. (2013) Historical perspectives in understanding traumatic brain injury and in situating disruption in CBF in the pathotrajectory of head trauma Cerebral Blood Flow, Metabolism, and Head Trauma: the Pathotrajectory of Traumatic Brain Injury. 1-27
Kreipke CW, Rafols JA. (2013) Cerebral blood flow, metabolism, and head trauma: The pathotrajectory of traumatic brain injury Cerebral Blood Flow, Metabolism, and Head Trauma: the Pathotrajectory of Traumatic Brain Injury. 1-169
Shenaq M, Kassem H, Peng C, et al. (2012) Neuronal damage and functional deficits are ameliorated by inhibition of aquaporin and HIF1α after traumatic brain injury (TBI). Journal of the Neurological Sciences. 323: 134-40
Ali A, Konakondla S, Zwagerman NT, et al. (2012) Glycerol accumulation in edema formation following diffuse traumatic brain injury. Neurological Research. 34: 462-8
Kane MJ, Angoa-Pérez M, Briggs DI, et al. (2012) A mouse model of human repetitive mild traumatic brain injury. Journal of Neuroscience Methods. 203: 41-9
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