Sarah E. Lutz, Ph.D.
Affiliations: | 2010 | Yeshiva University, New York, NY, United States |
Area:
neuro-immunologyGoogle:
"Sarah Lutz"Mean distance: 18.5 (cluster 46)
Parents
Sign in to add mentor| Celia Brosnan | grad student | 2010 | Yeshiva University | |
| (Loss of astrocyte connexin 43 and 30 modulates CNS myelin and inflammation.) | ||||
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Publications
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| Kalinin S, Meares GP, Lin SX, et al. (2019) Liver kinase B1 depletion from astrocytes worsens disease in a mouse model of multiple sclerosis. Glia |
| Lutz SE, Smith JR, Kim DH, et al. (2017) Caveolin1 Is Required for Th1 Cell Infiltration, but Not Tight Junction Remodeling, at the Blood-Brain Barrier in Autoimmune Neuroinflammation. Cell Reports. 21: 2104-2117 |
| Lim RG, Quan C, Reyes-Ortiz AM, et al. (2017) Huntington's Disease iPSC-Derived Brain Microvascular Endothelial Cells Reveal WNT-Mediated Angiogenic and Blood-Brain Barrier Deficits. Cell Reports. 19: 1365-1377 |
| Lengfeld JE, Lutz SE, Smith JR, et al. (2017) Endothelial Wnt/β-catenin signaling reduces immune cell infiltration in multiple sclerosis. Proceedings of the National Academy of Sciences of the United States of America |
| Velasquez S, Malik S, Lutz SE, et al. (2016) Pannexin1 Channels Are Required for Chemokine-Mediated Migration of CD4+ T Lymphocytes: Role in Inflammation and Experimental Autoimmune Encephalomyelitis. Journal of Immunology (Baltimore, Md. : 1950) |
| Chinnasamy P, Lutz SE, Riascos-Bernal DF, et al. (2015) Loss of Allograft Inflammatory Factor-1 Ameliorates Experimental Autoimmune Encephalomyelitis by Limiting Encephalitogenic CD4 T-Cell Expansion. Molecular Medicine (Cambridge, Mass.). 21: 233-41 |
| Lutz SE, González-Fernández E, Ventura JC, et al. (2013) Contribution of pannexin1 to experimental autoimmune encephalomyelitis. Plos One. 8: e66657 |
| Negoro H, Lutz SE, Liou LS, et al. (2013) Pannexin 1 involvement in bladder dysfunction in a multiple sclerosis model. Scientific Reports. 3: 2152 |
| Lutz SE, Raine CS, Brosnan CF. (2012) Loss of astrocyte connexins 43 and 30 does not significantly alter susceptibility or severity of acute experimental autoimmune encephalomyelitis in mice. Journal of Neuroimmunology. 245: 8-14 |
| Lutz SE, Zhao Y, Gulinello M, et al. (2009) Deletion of astrocyte connexins 43 and 30 leads to a dysmyelinating phenotype and hippocampal CA1 vacuolation. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 29: 7743-52 |