Hannah A. Hathaway, Ph.D.
Affiliations: | 2014 | Pharmacology | Georgetown University, Washington, DC |
Area:
Neuropharmacology, glutamate receptorsGoogle:
"Hannah Hathaway"Mean distance: 17.08 (cluster 6) | S | N | B | C | P |
Parents
Sign in to add mentorJarda T. Wroblewski | grad student | 2014 | Georgetown | |
(Function and signal transduction of metabotropic glutamate receptor 1 in cerebellar granule cells.) | ||||
Wendy B. Macklin | post-doc | University of Colorado, Denver |
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Publications
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Dahl KD, Almeida AR, Hathaway HA, et al. (2022) mTORC2 loss in oligodendrocyte progenitor cells results in regional hypomyelination in the central nervous system. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
Kohnke S, Buller S, Nuzzaci D, et al. (2021) Nutritional regulation of oligodendrocyte differentiation regulates perineuronal net remodeling in the median eminence. Cell Reports. 36: 109362 |
Gelb T, Pshenichkin S, Hathaway HA, et al. (2015) Atypical signaling of metabotropic glutamate receptor 1 in human melanoma cells. Biochemical Pharmacology. 98: 182-9 |
Hathaway HA, Pshenichkin S, Grajkowska E, et al. (2015) Pharmacological characterization of mGlu1 receptors in cerebellar granule cells reveals biased agonism. Neuropharmacology. 93: 199-208 |
Gelb T, Pshenichkin S, Rodriguez OC, et al. (2015) Metabotropic glutamate receptor 1 acts as a dependence receptor creating a requirement for glutamate to sustain the viability and growth of human melanomas. Oncogene. 34: 2711-20 |
Gelb T, Pshenichkin S, Hathaway HA, et al. (2015) Abstract A15: mGlu1 Receptors and downstream signal transduction proteins as therapeutic targets for the treatment of metastatic melanoma Cancer Research. 75 |
Gelb T, Hathaway HA, Wroblewski JT. (2014) Triple threat treatment: Exploiting the dependence receptor properties of metabotropic glutamate receptor 1 against melanoma. Molecular & Cellular Oncology. 1: e969163 |
DiRaddo JO, Miller EJ, Hathaway HA, et al. (2014) A real-time method for measuring cAMP production modulated by Gαi/o-coupled metabotropic glutamate receptors. The Journal of Pharmacology and Experimental Therapeutics. 349: 373-82 |
Emery AC, DiRaddo JO, Miller E, et al. (2012) Ligand bias at metabotropic glutamate 1a receptors: molecular determinants that distinguish β-arrestin-mediated from G protein-mediated signaling. Molecular Pharmacology. 82: 291-301 |