Amy S. Burnside, Ph.D.
Affiliations: | 2002 | University of Massachusetts, Amherst, Amherst, MA |
Area:
DevelopmentGoogle:
"Amy Burnside"Mean distance: 17.12 (cluster 11) | S | N | B | C | P |
Parents
Sign in to add mentor| Rolf O. Karlstrom | grad student | 2002 | U Mass Amherst | |
| (Reprogramming somatic gene expression through communication with blastomeres.) | ||||
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Publications
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| Wells AC, Daniels KA, Angelou CC, et al. (2017) Modulation of let-7 miRNAs controls the differentiation of effector CD8 T cells. Elife. 6 |
| Brown ML, Andrzejewski D, Burnside A, et al. (2016) Activin Enhances α- to β-cell Transdifferentiation As A Source For β-Cells In Male FSTL3 Knockout Mice. Endocrinology. en20151793 |
| Andrzejewski D, Brown ML, Ungerleider N, et al. (2015) Activins A and B Regulate Fate-Determining Gene Expression in Islet Cell Lines and Islet Cells From Male Mice. Endocrinology. 156: 2440-50 |
| Brown ML, Ungerleider N, Bonomi L, et al. (2014) Effects of activin A on survival, function and gene expression of pancreatic islets from non-diabetic and diabetic human donors. Islets. 6: e1017226 |
| Burnside AS, Collas P. (2002) Induction of Oct-3/4 expression in somatic cells by gap junction-mediated cAMP signaling from blastomeres. European Journal of Cell Biology. 81: 585-91 |
| Kasinathan P, Knott JG, Moreira PN, et al. (2001) Effect of fibroblast donor cell age and cell cycle on development of bovine nuclear transfer embryos in vitro. Biology of Reproduction. 64: 1487-93 |