Brian D. Hitt, Ph.D.
Affiliations: | 2012 | Neuroscience Institute Graduate Program | Northwestern University, Evanston, IL |
Area:
Alzheimer'sGoogle:
"Brian Hitt"Mean distance: 15.83 (cluster 11) | S | N | B | C | P |
Parents
Sign in to add mentor| Robert Vassar | grad student | 2012 | Northwestern | |
| (Seizure Susceptibility and Defective Outgrowth of Mossy Fiber Axons in BACE1-Deficient Mice.) | ||||
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Publications
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| Hitt B, Riordan SM, Kukreja L, et al. (2012) β-Site amyloid precursor protein (APP)-cleaving enzyme 1 (BACE1)-deficient mice exhibit a close homolog of L1 (CHL1) loss-of-function phenotype involving axon guidance defects. The Journal of Biological Chemistry. 287: 38408-25 |
| Hitt BD, Jaramillo TC, Chetkovich DM, et al. (2010) BACE1-/- mice exhibit seizure activity that does not correlate with sodium channel level or axonal localization. Molecular Neurodegeneration. 5: 31 |
| O'Connor T, Sadleir KR, Maus E, et al. (2008) Phosphorylation of the translation initiation factor eIF2alpha increases BACE1 levels and promotes amyloidogenesis. Neuron. 60: 988-1009 |
| Zhao J, Fu Y, Yasvoina M, et al. (2007) Beta-site amyloid precursor protein cleaving enzyme 1 levels become elevated in neurons around amyloid plaques: implications for Alzheimer's disease pathogenesis. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 27: 3639-49 |