Oscar Solis

Cajal Institute, CSIC, Madrid 
Parkinson disease, dyskinesia, dendritic spines
"Oscar Solis"
Mean distance: 14.97 (cluster 11)
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Suarez LM, Solis O, Sanz-Magro A, et al. (2020) Dopamine D1 Receptors Regulate Spines in Striatal Direct-Pathway and Indirect-Pathway Neurons. Movement Disorders : Official Journal of the Movement Disorder Society
Solís O, García-Sanz P, Martín AB, et al. (2019) Behavioral sensitization and cellular responses to psychostimulants are reduced in D2R knockout mice. Addiction Biology. e12840
Keifman E, Ruiz de Diego I, Pafundo DE, et al. (2019) Optostimulation of striatonigral terminals in substantia nigra induces dyskinesia that increases after L-DOPA in a mouse model of Parkinson's disease. British Journal of Pharmacology
Ruiz-DeDiego I, Fasano S, Solís O, et al. (2018) Genetic enhancement of Ras-ERK pathway does not aggravate L-DOPA-induced dyskinesia in mice but prevents the decrease induced by lovastatin. Scientific Reports. 8: 15381
García-Montes JR, Solís O, Enríquez-Traba J, et al. (2018) Genetic Knockdown of mGluR5 in Striatal D1R-Containing Neurons Attenuates L-DOPA-Induced Dyskinesia in Aphakia Mice. Molecular Neurobiology
Solís O, Moratalla R. (2018) Dopamine receptors: homomeric and heteromeric complexes in L-DOPA-induced dyskinesia. Journal of Neural Transmission (Vienna, Austria : 1996)
Suarez LM, Solis O, Aguado C, et al. (2016) L-DOPA Oppositely Regulates Synaptic Strength and Spine Morphology in D1 and D2 Striatal Projection Neurons in Dyskinesia. Cerebral Cortex (New York, N.Y. : 1991)
Solís O, García-Sanz P, Herranz AS, et al. (2016) L-DOPA Reverses the Increased Free Amino Acids Tissue Levels Induced by Dopamine Depletion and Rises GABA and Tyrosine in the Striatum. Neurotoxicity Research
Rodríguez-Traver E, Solís O, Díaz-Guerra E, et al. (2015) Role of Nurr1 in the Generation and Differentiation of Dopaminergic Neurons from Stem Cells. Neurotoxicity Research
Solís O, Garcia-Montes JR, González-Granillo A, et al. (2015) Dopamine D3 Receptor Modulates l-DOPA-Induced Dyskinesia by Targeting D1 Receptor-Mediated Striatal Signaling. Cerebral Cortex (New York, N.Y. : 1991)
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