Kai Kaila

Affiliations: 
University of Helsinki, Helsingfors, Finland 
Area:
development, GABAergic transmission, pH, epilepsy
Website:
http://www.helsinki.fi/neurobiology/
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"Kai Kaila"
Mean distance: 14.53 (cluster 6)
 
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Publications

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Remzső G, Németh J, Varga V, et al. (2020) Brain interstitial pH changes in the subacute phase of hypoxic-ischemic encephalopathy in newborn pigs. Plos One. 15: e0233851
Zions M, Meehan EF, Kress ME, et al. (2020) Nest Carbon Dioxide Masks GABA-Dependent Seizure Susceptibility in the Naked Mole-Rat. Current Biology : Cb
Pospelov AS, Puskarjov M, Kaila K, et al. (2020) Endogenous brain-sparing responses in brain pH and P in a rodent model of birth asphyxia. Acta Physiologica (Oxford, England). e13467
Mavrovic M, Uvarov P, Delpire E, et al. (2020) Loss of non-canonical KCC2 functions promotes developmental apoptosis of cortical projection neurons. Embo Reports. e48880
Spoljaric I, Spoljaric A, Mavrovic M, et al. (2019) KCC2-Mediated Cl Extrusion Modulates Spontaneous Hippocampal Network Events in Perinatal Rats and Mice. Cell Reports. 26: 1073-1081.e3
Brandt C, Seja P, Töllner K, et al. (2018) Corrigendum to "Bumepamine, a brain-permeant benzylamine derivative of bumetanide, does not inhibit NKCC1 but is more potent to enhance phenobarbital's antiseizure efficacy" [Neuropharmacology 143 (2018) 186-204]. Neuropharmacology
Brandt C, Seja P, Töllner K, et al. (2018) Bumepamine, a brain-permeant benzylamine derivative of bumetanide, does not inhibit NKCC1 but is more potent to enhance phenobarbital's anti-seizure efficacy. Neuropharmacology
Summanen M, Bäck S, Voipio J, et al. (2018) Surge of Peripheral Arginine Vasopressin in a Rat Model of Birth Asphyxia. Frontiers in Cellular Neuroscience. 12: 2
Spoljaric A, Seja P, Spoljaric I, et al. (2017) Vasopressin excites interneurons to suppress hippocampal network activity across a broad span of brain maturity at birth. Proceedings of the National Academy of Sciences of the United States of America. 114: E10819-E10828
Kelen D, Andorka C, Szabó M, et al. (2017) Serum copeptin and neuron specific enolase are markers of neonatal distress and long-term neurodevelopmental outcome. Plos One. 12: e0184593
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