Gavriel David
Affiliations: | University of Miami Miller School of Medicine, Miami, FL, United States |
Area:
Physiology and pathology of Calcium handling in motor axons and terminalsWebsite:
http://chroma.med.miami.edu/physiol/faculty-david_g.htmGoogle:
"Gavriel David"Mean distance: 14.38 (cluster 11) | S | N | B | C | P |
Parents
Sign in to add mentor| Yoel Yaari | grad student | 1982-1986 | Hebrew University |
| Ellen F. Barrett | post-doc | 1989-1991 | University of Miami Miller School of Medicine |
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Publications
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| Zhang Z, David G. (2015) Stimulation-induced Ca(2+) influx at nodes of Ranvier in mouse peripheral motor axons. The Journal of Physiology |
| Barrett EF, Barrett JN, David G. (2014) Dysfunctional mitochondrial Ca(2+) handling in mutant SOD1 mouse models of fALS: integration of findings from motor neuron somata and motor terminals. Frontiers in Cellular Neuroscience. 8: 184 |
| Nguyen KT, Zhang Z, Barrett EF, et al. (2012) Morphological and functional changes in innervation of a fast forelimb muscle in SOD1-G85R mice. Neurobiology of Disease. 48: 399-408 |
| Talbot JD, David G, Barrett EF, et al. (2012) Calcium dependence of damage to mouse motor nerve terminals following oxygen/glucose deprivation. Experimental Neurology. 234: 95-104 |
| Barrett EF, Barrett JN, David G. (2011) Mitochondria in motor nerve terminals: function in health and in mutant superoxide dismutase 1 mouse models of familial ALS. Journal of Bioenergetics and Biomembranes. 43: 581-6 |
| Nguyen KT, Barrett JN, GarcÃa-Chacón L, et al. (2011) Repetitive nerve stimulation transiently opens the mitochondrial permeability transition pore in motor nerve terminals of symptomatic mutant SOD1 mice. Neurobiology of Disease. 42: 381-90 |
| Zhang Z, Nguyen KT, Barrett EF, et al. (2010) Vesicular ATPase inserted into the plasma membrane of motor terminals by exocytosis alkalinizes cytosolic pH and facilitates endocytosis. Neuron. 68: 1097-108 |
| Nguyen KT, GarcÃa-Chacón LE, Barrett JN, et al. (2009) The Psi(m) depolarization that accompanies mitochondrial Ca2+ uptake is greater in mutant SOD1 than in wild-type mouse motor terminals. Proceedings of the National Academy of Sciences of the United States of America. 106: 2007-11 |
| Talbot JD, Barrett JN, Barrett EF, et al. (2008) Rapid, stimulation-induced reduction of C12-resorufin in motor nerve terminals: linkage to mitochondrial metabolism. Journal of Neurochemistry. 105: 807-19 |
| David G, Nguyen K, Barrett EF. (2007) Early vulnerability to ischemia/reperfusion injury in motor terminals innervating fast muscles of SOD1-G93A mice. Experimental Neurology. 204: 411-20 |