Mark Ansorge, Phd

Affiliations: 
Columbia University, New York, NY 
Area:
models of Psychiatric Disorders, Neurobiology of Learning and Memory, Synapses and Circuits
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"Mark Ansorge"
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Publications

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Demireva EY, Suri D, Morelli E, et al. (2018) 5-HT2C receptor blockade reverses SSRI-associated basal ganglia dysfunction and potentiates therapeutic efficacy. Molecular Psychiatry
Teixeira CM, Rosen ZB, Suri D, et al. (2018) Hippocampal 5-HT Input Regulates Memory Formation and Schaffer Collateral Excitation. Neuron
Garcia-Garcia AL, Canetta S, Stujenske JM, et al. (2017) Serotonin inputs to the dorsal BNST modulate anxiety in a 5-HT receptor-dependent manner. Molecular Psychiatry. 23: 1990-1997
Gingrich JA, Malm H, Ansorge MS, et al. (2017) New Insights into How Serotonin Selective Reuptake Inhibitors Shape the Developing Brain. Birth Defects Research. 109: 924-932
Bansal R, Peterson BS, Gingrich J, et al. (2016) Serotonin signaling modulates the effects of familial risk for depression on cortical thickness. Psychiatry Research
Rebello TJ, Yu Q, Goodfellow NM, et al. (2014) Postnatal day 2 to 11 constitutes a 5-HT-sensitive period impacting adult mPFC function. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 12379-93
Goodfellow NM, Sargin D, Ansorge MS, et al. (2014) Mice with compromised 5-HTT function lack phosphotyrosine-mediated inhibitory control over prefrontal 5-HT responses. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 6107-11
Vitalis T, Ansorge MS, Dayer AG. (2013) Serotonin homeostasis and serotonin receptors as actors of cortical construction: special attention to the 5-HT3A and 5-HT6 receptor subtypes. Frontiers in Cellular Neuroscience. 7: 93
Morelli E, Moore H, Rebello TJ, et al. (2011) Chronic 5-HT transporter blockade reduces DA signaling to elicit basal ganglia dysfunction. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 31: 15742-50
Muller JM, Morelli E, Ansorge M, et al. (2011) Serotonin transporter deficient mice are vulnerable to escape deficits following inescapable shocks. Genes, Brain, and Behavior. 10: 166-75
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