Dimitri Krainc

Affiliations: 
Northwestern University, Evanston, IL 
Area:
Cell Biology, Disease, Molecular Neuroscience, Neurobiology of Disease
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"Dimitri Krainc"
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Kim MJ, Kim S, Reinheckel T, et al. (2024) Inhibition of cysteine protease cathepsin Lincreases the level and activity of lysosomal glucocerebrosidase. Jci Insight. 9
Brooker SM, Naylor GE, Krainc D. (2024) Cell biology of Parkinson's disease: Mechanisms of synaptic, lysosomal, and mitochondrial dysfunction. Current Opinion in Neurobiology. 85: 102841
Song P, Peng W, Sauve V, et al. (2023) Parkinson's disease-linked parkin mutation disrupts recycling of synaptic vesicles in human dopaminergic neurons. Neuron
Peng W, Schröder LF, Song P, et al. (2023) Parkin regulates amino acid homeostasis at mitochondria-lysosome (M/L) contact sites in Parkinson's disease. Science Advances. 9: eadh3347
Chatterjee D, Krainc D. (2023) Mechanisms of Glucocerebrosidase Dysfunction in Parkinson's Disease. Journal of Molecular Biology. 168023
Wong YC, Kim S, Cisneros J, et al. (2022) Mid51/Fis1 mitochondrial oligomerization complex drives lysosomal untethering and network dynamics. The Journal of Cell Biology. 221
Kim S, Coukos R, Gao F, et al. (2022) Dysregulation of organelle membrane contact sites in neurological diseases. Neuron
Burbulla LF, Zheng J, Song P, et al. (2021) Direct targeting of wild-type glucocerebrosidase by antipsychotic quetiapine improves pathogenic phenotypes in Parkinson's disease models. Jci Insight. 6
Brooker SM, Krainc D. (2021) Glucocerebrosidase dysfunction in neurodegenerative disease. Essays in Biochemistry
Kim S, Wong YC, Gao F, et al. (2021) Dysregulation of mitochondria-lysosome contacts by GBA1 dysfunction in dopaminergic neuronal models of Parkinson's disease. Nature Communications. 12: 1807
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