Alexandra M. Nicholson, Ph.D.
Affiliations: | 2010 | Integrated Graduate Program in the Life Sciences | Northwestern University, Evanston, IL |
Area:
Cell Biology, Molecular Neuroscience, Neurobiology of DiseaseGoogle:
"Alexandra Nicholson"Mean distance: 35622
Parents
Sign in to add mentor| Adriana Ferreira | grad student | 2010 | Northwestern | |
| (Membrane cholesterol in the pathogenesis of Alzheimer's disease.) | ||||
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Publications
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| Zhou X, Nicholson AM, Ren Y, et al. (2020) Loss of TMEM106B leads to myelination deficits: implications for frontotemporal dementia treatment strategies. Brain : a Journal of Neurology |
| Arrant AE, Nicholson AM, Zhou X, et al. (2018) Partial Tmem106b reduction does not correct abnormalities due to progranulin haploinsufficiency. Molecular Neurodegeneration. 13: 32 |
| Nicholson AM, Zhou X, Perkerson RB, et al. (2018) Loss of Tmem106b is unable to ameliorate frontotemporal dementia-like phenotypes in an AAV mouse model of C9ORF72-repeat induced toxicity. Acta Neuropathologica Communications. 6: 42 |
| Hirsch-Reinshagen V, Pottier C, Nicholson AM, et al. (2017) Clinical and neuropathological features of ALS/FTD with TIA1 mutations. Acta Neuropathologica Communications. 5: 96 |
| Mackenzie IR, Nicholson AM, Sarkar M, et al. (2017) TIA1 Mutations in Amyotrophic Lateral Sclerosis and Frontotemporal Dementia Promote Phase Separation and Alter Stress Granule Dynamics. Neuron. 95: 808-816.e9 |
| Molgaard S, Demontis D, Nicholson AM, et al. (2016) Soluble sortilin is present in excess and positively correlates with progranulin in CSF of aging individuals. Experimental Gerontology |
| Nicholson AM, Rademakers R. (2016) What we know about TMEM106B in neurodegeneration. Acta Neuropathologica |
| Nicholson AM, Finch NA, Almeida M, et al. (2016) Prosaposin is a regulator of progranulin levels and oligomerization. Nature Communications. 7: 11992 |
| Dickson DW, Rademakers R, Nicholson AM, et al. (2015) The TMEM106B locus and TDP-43 pathology in older persons without FTLD. Neurology. 85: 1354-5 |
| Pottier C, Bieniek KF, Finch N, et al. (2015) Whole-genome sequencing reveals important role for TBK1 and OPTN mutations in frontotemporal lobar degeneration without motor neuron disease. Acta Neuropathologica. 130: 77-92 |