Todd N. Eagar, Ph.D.

Affiliations: 
2002 Northwestern University, Evanston, IL 
Area:
Molecular Neuroscience, Neurobiology of Disease, Neuroimmunology
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"Todd Eagar"
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Stephen Miller grad student 2002 Northwestern
 (Regulation of T cell tolerance by antigen -coupled splenocyte injection.)
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Publications

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Cummings M, Arumanayagam ACS, Zhao P, et al. (2018) Presenilin1 regulates Th1 and Th17 effector responses but is not required for experimental autoimmune encephalomyelitis. Plos One. 13: e0200752
Arellano B, Hussain R, Miller-Little WA, et al. (2016) A Single Amino Acid Substitution Prevents Recognition of a Dominant Human Aquaporin-4 Determinant in the Context of HLA-DRB1*03:01 by a Murine TCR. Plos One. 11: e0152720
Raphael I, Nalawade S, Eagar TN, et al. (2015) T cell subsets and their signature cytokines in autoimmune and inflammatory diseases. Cytokine. 74: 5-17
Hussain RZ, Hayardeny L, Cravens PC, et al. (2014) Immune surveillance of the central nervous system in multiple sclerosis--relevance for therapy and experimental models. Journal of Neuroimmunology. 276: 9-17
Castro-Rojas C, Deason K, Hussain RZ, et al. (2014) Testing effects of glatiramer acetate and fingolimod in an infectious model of CNS immune surveillance. Journal of Neuroimmunology. 276: 232-5
Lambracht-Washington D, Qu BX, Fu M, et al. (2013) A peptide prime-DNA boost immunization protocol provides significant benefits as a new generation Aβ42 DNA vaccine for Alzheimer disease. Journal of Neuroimmunology. 254: 63-8
Eagar TN, Miller SD. (2012) Helper T-cell subsets and control of the inflammatory response Clinical Immunology: Principles and Practice: Fourth Edition. 203-214
Cravens PD, Hussain RZ, Zacharias TE, et al. (2011) Lymph node-derived donor encephalitogenic CD4+ T cells in C57BL/6 mice adoptive transfer experimental autoimmune encephalomyelitis highly express GM-CSF and T-bet. Journal of Neuroinflammation. 8: 73
German DC, Eagar T, Sonsalla PK. (2011) Parkinson's Disease: A Role for the Immune System. Current Molecular Pharmacology
Lambracht-Washington D, Qu BX, Fu M, et al. (2011) DNA immunization against amyloid beta 42 has high potential as safe therapy for Alzheimer's disease as it diminishes antigen-specific Th1 and Th17 cell proliferation. Cellular and Molecular Neurobiology. 31: 867-74
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