Hui Wang
Affiliations: | University of Maryland, College Park, College Park, MD |
Area:
Alzheimer's diseaseGoogle:
"Hui Wang"Mean distance: 14.98 (cluster 6) | S | N | B | C | P |
Cross-listing: Alzheimer's Tree
Parents
Sign in to add mentor| Hey-Kyoung Lee | grad student | 2007-2012 | University of Maryland | |
| (Circuit analysis of synaptic dysfunctions in the CA3 area of BACE1 knockout mice.) | ||||
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Publications
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| Wang H, Fu J, Xu X, et al. (2021) Rapamycin activates mitophagy and alleviates cognitive and synaptic plasticity deficits in a mouse model of Alzheimer's disease. The Journals of Gerontology. Series a, Biological Sciences and Medical Sciences |
| Chokshi V, Gao M, Grier BD, et al. (2019) Input-Specific Metaplasticity in the Visual Cortex Requires Homer1a-Mediated mGluR5 Signaling. Neuron |
| Wang H, Ardiles AO, Yang S, et al. (2016) Metabotropic Glutamate Receptors Induce a Form of LTP Controlled by Translation and Arc Signaling in the Hippocampus. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 36: 1723-9 |
| Gao M, Maynard KR, Chokshi V, et al. (2014) Rebound potentiation of inhibition in juvenile visual cortex requires vision-induced BDNF expression. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 10770-9 |
| Wang H, Megill A, Wong PC, et al. (2014) Postsynaptic target specific synaptic dysfunctions in the CA3 area of BACE1 knockout mice. Plos One. 9: e92279 |
| Petrus E, Isaiah A, Jones AP, et al. (2014) Crossmodal induction of thalamocortical potentiation leads to enhanced information processing in the auditory cortex. Neuron. 81: 664-73 |
| LeGates TA, Altimus CM, Wang H, et al. (2012) Aberrant light directly impairs mood and learning through melanopsin-expressing neurons. Nature. 491: 594-8 |
| Wang H, Megill A, He K, et al. (2012) Consequences of inhibiting amyloid precursor protein processing enzymes on synaptic function and plasticity. Neural Plasticity. 2012: 272374 |
| Wang H, Song L, Lee A, et al. (2010) Mossy fiber long-term potentiation deficits in BACE1 knock-outs can be rescued by activation of alpha7 nicotinic acetylcholine receptors. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 30: 13808-13 |
| Wang H, Song L, Laird F, et al. (2008) BACE1 knock-outs display deficits in activity-dependent potentiation of synaptic transmission at mossy fiber to CA3 synapses in the hippocampus. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 28: 8677-81 |