M. J. Pinter

University of Göteborg, Gothenburg, Västra Götalands län, Sweden 
"M. Pinter"
Mean distance: 15.35 (cluster 6)
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Carrasco DI, Bahr BA, Seburn KL, et al. (2016) Abnormal response of distal Schwann cells to denervation in a mouse model of motor neuron disease. Experimental Neurology
Wang X, Pinter MJ, Rich MM. (2016) Reversible Recruitment of a Homeostatic Reserve Pool of Synaptic Vesicles Underlies Rapid Homeostatic Plasticity of Quantal Content. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 36: 828-36
Carrasco DI, Seburn KL, Pinter MJ. (2015) Altered terminal Schwann cell morphology precedes denervation in SOD1 mice. Experimental Neurology
Novak KR, Norman J, Mitchell JR, et al. (2015) Sodium channel slow inactivation as a therapeutic target for myotonia congenita. Annals of Neurology. 77: 320-32
Carrasco DI, Bichler EK, Rich MM, et al. (2012) Motor terminal degeneration unaffected by activity changes in SOD1(G93A) mice; a possible role for glycolysis. Neurobiology of Disease. 48: 132-40
Bullinger KL, Nardelli P, Pinter MJ, et al. (2011) Permanent central synaptic disconnection of proprioceptors after nerve injury and regeneration. II. Loss of functional connectivity with motoneurons. Journal of Neurophysiology. 106: 2471-85
Prather JF, Nardelli P, Nakanishi ST, et al. (2011) Recovery of proprioceptive feedback from nerve crush. The Journal of Physiology. 589: 4935-47
Carrasco DI, Bichler EK, Seburn KL, et al. (2010) Nerve terminal degeneration is independent of muscle fiber genotype in SOD1 mice. Plos One. 5: e9802
Wang X, Pinter MJ, Rich MM. (2010) Ca2+ dependence of the binomial parameters p and n at the mouse neuromuscular junction. Journal of Neurophysiology. 103: 659-66
Filatov GN, Pinter MJ, Rich MM. (2009) Role of Ca(2+) in injury-induced changes in sodium current in rat skeletal muscle. American Journal of Physiology. Cell Physiology. 297: C352-9
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