Lloyd Greene

Columbia University, New York, NY 
neuronal death, neural development
"Lloyd Greene"
Mean distance: 14.44 (cluster 11)
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Sun X, Jefferson P, Zhou Q, et al. (2019) Dominant-Negative ATF5 Compromises Cancer Cell Survival by Targeting CEBPB and CEBPD. Molecular Cancer Research : McR
Sun X, Angelastro JM, Merino D, et al. (2019) Dominant-negative ATF5 rapidly depletes survivin in tumor cells. Cell Death & Disease. 10: 709
Corona C, Pasini S, Liu J, et al. (2018) Activating Transcription Factor 4 (ATF4) regulates neuronal activity by controlling GABAR trafficking. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Liu J, Amar F, Corona C, et al. (2018) Brain-Derived Neurotrophic Factor Elevates Activating Transcription Factor 4 (ATF4) in Neurons and Promotes ATF4-Dependent Induction of. Frontiers in Molecular Neuroscience. 11: 62
Biswas SC, Sanphui P, Chatterjee N, et al. (2017) Cdc25A phosphatase: a key cell cycle protein that regulates neuron death in disease and development. Cell Death & Disease. 8: e2692
Chatterjee N, Sanphui P, Kemeny S, et al. (2016) Role and regulation of Cdc25A phosphatase in neuron death induced by NGF deprivation or β-amyloid. Cell Death Discovery. 2: 16083
Pasini S, Liu J, Corona C, et al. (2016) Activating Transcription Factor 4 (ATF4) modulates Rho GTPase levels and function via regulation of RhoGDIα. Scientific Reports. 6: 36952
Karpel-Massler G, Horst BA, Shu C, et al. (2016) A synthetic cell-penetrating dominant-negative ATF5 peptide exerts anti-cancer activity against a broad spectrum of treatment resistant cancers. Clinical Cancer Research : An Official Journal of the American Association For Cancer Research
Cates CC, Arias AD, Nakayama Wong LS, et al. (2016) Regression/Eradication of gliomas in mice by a systemically-deliverable ATF5 dominant-negative peptide. Oncotarget
Aimé P, Sun X, Zareen N, et al. (2015) Trib3 Is Elevated in Parkinson's Disease and Mediates Death in Parkinson's Disease Models. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 35: 10731-49
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