Vytas K. Verselis

Albert Einstein College of Medicine, New York, New York, United States 
gap junction
"Vytas Verselis"
Mean distance: 15.37 (cluster 16)
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Spray DC, Verselis VK, Bennett MVL. (2019) Introduction to Connexins and Pannexins in the Healthy and Diseased Nervous System with Thanks to Felikas Bukauskas. Neuroscience Letters. 695: 1-3
Sanchez HA, Verselis VK. (2014) Aberrant Cx26 hemichannels and keratitis-ichthyosis-deafness syndrome: insights into syndromic hearing loss. Frontiers in Cellular Neuroscience. 8: 354
Sanchez HA, Bienkowski R, Slavi N, et al. (2014) Altered inhibition of Cx26 hemichannels by pH and Zn2+ in the A40V mutation associated with keratitis-ichthyosis-deafness syndrome. The Journal of Biological Chemistry. 289: 21519-32
Sanchez HA, Villone K, Srinivas M, et al. (2013) The D50N mutation and syndromic deafness: altered Cx26 hemichannel properties caused by effects on the pore and intersubunit interactions. The Journal of General Physiology. 142: 3-22
Verselis VK, Srinivas M. (2013) Connexin channel modulators and their mechanisms of action. Neuropharmacology. 75: 517-24
Kronengold J, Srinivas M, Verselis VK. (2012) The N-terminal half of the connexin protein contains the core elements of the pore and voltage gates. The Journal of Membrane Biology. 245: 453-63
Rubinos C, Sánchez HA, Verselis VK, et al. (2012) Mechanism of inhibition of connexin channels by the quinine derivative N-benzylquininium. The Journal of General Physiology. 139: 69-82
Sánchez HA, Mese G, Srinivas M, et al. (2010) Differentially altered Ca2+ regulation and Ca2+ permeability in Cx26 hemichannels formed by the A40V and G45E mutations that cause keratitis ichthyosis deafness syndrome. The Journal of General Physiology. 136: 47-62
Sánchez HA, Orellana JA, Verselis VK, et al. (2009) Metabolic inhibition increases activity of connexin-32 hemichannels permeable to Ca2+ in transfected HeLa cells. American Journal of Physiology. Cell Physiology. 297: C665-78
Tang Q, Dowd TL, Verselis VK, et al. (2009) Conformational changes in a pore-forming region underlie voltage-dependent "loop gating" of an unapposed connexin hemichannel. The Journal of General Physiology. 133: 555-70
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