Steven Stasheff
Affiliations: | University of Iowa, Iowa City, IA |
Area:
Visual systemWebsite:
http://www.uihealthcare.com/depts/med/pediatrics/pedsmds/stasheff_research.htmlGoogle:
"Steven Stasheff"Mean distance: 15.57 (cluster 6) | S | N | B | C | P |
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Publications
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Thompson S, Blodi FR, Larson DR, et al. (2019) The Efemp1R345W Macular Dystrophy Mutation Causes Amplified Circadian and Photophobic Responses to Light in Mice. Investigative Ophthalmology & Visual Science. 60: 2110-2117 |
Thompson S, Blodi FR, Lee S, et al. (2014) Photoreceptor cells with profound structural deficits can support useful vision in mice. Investigative Ophthalmology & Visual Science. 55: 1859-66 |
Clark S, Stasheff S, Lewis DV, et al. (2012) The NMDA receptor in epilepsy The Nmda Receptor |
Thompson S, Stasheff SF, Hernandez J, et al. (2011) Different inner retinal pathways mediate rod-cone input in irradiance detection for the pupillary light reflex and regulation of behavioral state in mice. Investigative Ophthalmology & Visual Science. 52: 618-23 |
Burack MA, Stasheff SF, Wilson WA. (1995) Selective suppression of in vitro electrographic seizures by low-dose tetrodotoxin: a novel anticonvulsant effect. Epilepsy Research. 22: 115-26 |
Stasheff SF, Hines M, Wilson WA. (1993) Axon terminal hyperexcitability associated with epileptogenesis in vitro. I. Origin of ectopic spikes. Journal of Neurophysiology. 70: 961-75 |
Stasheff SF, Mott DD, Wilson WA. (1993) Axon terminal hyperexcitability associated with epileptogenesis in vitro. II. Pharmacological regulation by NMDA and GABAA receptors. Journal of Neurophysiology. 70: 976-84 |
Stasheff SF, Wilson WA. (1992) Axon terminal hyperexcitability seen in epileptogenesis in vitro. Ion Channels. 3: 137-57 |
Wilson WA, Stasheff S, Swartzwelder S, et al. (1992) The role of NMDA receptors in in vitro epileptogenesis. Epilepsy Research. Supplement. 8: 157-65; discussion 1 |
Stasheff SF, Wilson WA. (1990) Increased ectopic action potential generation accompanies epileptogenesis in vitro. Neuroscience Letters. 111: 144-50 |