Seth A. Ament

Affiliations: 
2016- Institute for Genome Sciences University of Maryland School of Medicine, Baltimore, MD, United States 
Area:
behavioral & physiological genomics
Website:
http://www.medschool.umaryland.edu/profiles/Seth-Ament/
Google:
"Seth Ament"
Mean distance: 17.33 (cluster 43)
 
SNBCP

Parents

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Hans A. Hofmann research assistant 2001-2003 Harvard
Gene E. Robinson grad student 2004-2010 UIUC
 (Nutrition, hormones, transcriptional regulatory networks and division of labor in honey bee colonies.)
Nathan Daniel Price post-doc Institute for Systems Biology (Computational Biology Tree)
Leroy Hood post-doc 2012-2016 Institute for Systems Biology (Computational Biology Tree)
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Publications

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Malaiya S, Cortes-Gutierrez M, Herb BR, et al. (2021) Single-Nucleus RNA-Seq Reveals Dysregulation of Striatal Cell Identity Due to Huntington's Disease Mutations. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Funk CC, Casella AM, Jung S, et al. (2020) Atlas of Transcription Factor Binding Sites from ENCODE DNase Hypersensitivity Data across 27 Tissue Types. Cell Reports. 32: 108029
Pearl JR, Colantuoni C, Bergey DE, et al. (2019) Genome-Scale Transcriptional Regulatory Network Models of Psychiatric and Neurodegenerative Disorders. Cell Systems
Budde M, Friedrichs S, Alliey-Rodriguez N, et al. (2018) Efficient region-based test strategy uncovers genetic risk factors for functional outcome in bipolar disorder. European Neuropsychopharmacology : the Journal of the European College of Neuropsychopharmacology
Ament SA, Pearl JR, Cantle JP, et al. (2018) Transcriptional regulatory networks underlying gene expression changes in Huntington's disease. Molecular Systems Biology. 14: e7435
Coffey SR, Bragg RM, Minnig S, et al. (2017) Peripheral huntingtin silencing does not ameliorate central signs of disease in the B6.HttQ111/+ mouse model of Huntington's disease. Plos One. 12: e0175968
Bragg RM, Coffey SR, Weston RM, et al. (2017) Corrigendum: Motivational, proteostatic and transcriptional deficits precede synapse loss, gliosis and neurodegeneration in the B6.Htt(Q111/+) model of Huntington's disease. Scientific Reports. 7: 44960
Ament SA, Pearl JR, Grindeland A, et al. (2017) High resolution time-course mapping of early transcriptomic, molecular and cellular phenotypes in Huntington's disease CAG knock-in mice across multiple genetic backgrounds. Human Molecular Genetics
Bragg RM, Coffey SR, Weston RM, et al. (2017) Motivational, proteostatic and transcriptional deficits precede synapse loss, gliosis and neurodegeneration in the B6.Htt(Q111/+) model of Huntington's disease. Scientific Reports. 7: 41570
Wheeler MM, Ament SA, Rodriguez-Zas SL, et al. (2015) Diet and endocrine effects on behavioral maturation-related gene expression in the pars intercerebralis of the honey bee brain. The Journal of Experimental Biology
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