Donald A. Simone

Affiliations: 
Neuroscience University of Minnesota, Twin Cities, Minneapolis, MN 
Area:
Neuroscience Biology, Physiology Biology
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"Donald Simone"
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Children

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Darryl T. Hamamoto grad student 2000 UMN
Lisa M. Johanek grad student 2004 UMN
Walter R. Bowles grad student 2009 UMN
Carl R. Potenzieri grad student 2009 UMN
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Publications

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Banik RK, Sia T, Ibrahim MM, et al. (2023) Increases in local skin temperature correlate with spontaneous foot lifting and heat hyperalgesia in both incisional inflammatory models of pain. Pain Reports. 8: e1097
Rogness VM, Juliette J, Khasabova IA, et al. (2023) Descending facilitation of nociceptive transmission from the rostral ventromedial medulla contributes to hyperalgesia in mice with sickle cell disease. Neuroscience
Khasabova IA, Khasabov SG, Johns M, et al. (2023) Exosome-associated lysophosphatidic acid signaling contributes to cancer pain. Pain
Cataldo G, Lunzer MM, Akgün E, et al. (2023) MMG22 potently blocks hyperalgesia in cisplatin-treated mice. Neuroscience
Khasabova IA, Seybold VS, Simone DA. (2021) The role of PPARγ in chemotherapy-evoked pain. Neuroscience Letters. 135845
Khasabov SG, Rogness VM, Beeson MB, et al. (2021) The nAChR chaperone TMEM35a (NACHO) contributes to the development of hyperalgesia in mice. Neuroscience
Tran PV, Johns ME, McAdams B, et al. (2020) Global transcriptome analysis of rat dorsal root ganglia to identify molecular pathways involved in incisional pain. Molecular Pain. 16: 1744806920956480
Uhelski ML, Bruce D, Speltz R, et al. (2020) Topical Application of Loperamide/Oxymorphindole, Mu and Delta Opioid Receptor Agonists, Reduces Sensitization of C-fiber Nociceptors that Possess Na1.8. Neuroscience
Khasabova IA, Golovko MY, Golovko SA, et al. (2020) Intrathecal Administration of Resolvin D1 and E1 Decreases Hyperalgesia in Mice with Bone Cancer Pain: Involvement of Endocannabinoid Signaling. Prostaglandins & Other Lipid Mediators. 106479
Speltz R, Lunzer MM, Shueb SS, et al. (2020) The bivalent ligand, MMG22, reduces neuropathic pain after nerve injury without the side effects of traditional opioids. Pain
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