Tracy K. McIntosh

Media Neuroconsultants 
"Tracy McIntosh"
Mean distance: 17.55 (cluster 52)
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Lifshitz J, Rowe RK, Griffiths DR, et al. (2016) Clinical relevance of midline fluid percussion brain injury: Acute deficits, chronic morbidities and the utility of biomarkers. Brain Injury. 1-9
Tomasevic G, Laurer HL, Mattiasson G, et al. (2012) Delayed neuromotor recovery and increased memory acquisition dysfunction following experimental brain trauma in mice lacking the DNA repair gene XPA. Journal of Neurosurgery. 116: 1368-78
Tomasevic G, Raghupathi R, Scherbel U, et al. (2010) Deletion of the p53 tumor suppressor gene improves neuromotor function but does not attenuate regional neuronal cell loss following experimental brain trauma in mice. Journal of Neuroscience Research. 88: 3414-23
Hånell A, Clausen F, Björk M, et al. (2010) Genetic deletion and pharmacological inhibition of Nogo-66 receptor impairs cognitive outcome after traumatic brain injury in mice. Journal of Neurotrauma. 27: 1297-309
Marklund N, Morales D, Clausen F, et al. (2009) Functional outcome is impaired following traumatic brain injury in aging Nogo-A/B-deficient mice. Neuroscience. 163: 540-51
Conte V, Raghupathi R, Watson DJ, et al. (2008) TrkB gene transfer does not alter hippocampal neuronal loss and cognitive deficits following traumatic brain injury in mice. Restorative Neurology and Neuroscience. 26: 45-56
Marklund N, Bareyre FM, Royo NC, et al. (2007) Cognitive outcome following brain injury and treatment with an inhibitor of Nogo-A in association with an attenuated downregulation of hippocampal growth-associated protein-43 expression. Journal of Neurosurgery. 107: 844-53
Keck CA, Thompson HJ, Pitkänen A, et al. (2007) The novel antiepileptic agent RWJ-333369-A, but not its analog RWJ-333369, reduces regional cerebral edema without affecting neurobehavioral outcome or cell death following experimental traumatic brain injury. Restorative Neurology and Neuroscience. 25: 77-90
Thompson HJ, Marklund N, LeBold DG, et al. (2006) Tissue sparing and functional recovery following experimental traumatic brain injury is provided by treatment with an anti-myelin-associated glycoprotein antibody. The European Journal of Neuroscience. 24: 3063-72
Johnson VE, Murray L, Raghupathi R, et al. (2006) No evidence for the presence of apolipoprotein epsilon4, interleukin-lalpha allele 2 and interleukin-1beta allele 2 cause an increase in programmed cell death following traumatic brain injury in humans. Clinical Neuropathology. 25: 255-64
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