Brian D. Perkins

2004-2012 Biology Texas A & M University, College Station, TX, United States 
 2012- Cole Eye Institute The Cleveland Clinic, Cleveland, OH, United States 
retinal development
"Brian Perkins"
Mean distance: 14.19 (cluster 11)
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Perkins BD, Lobo GP, Kondkar AA, et al. (2023) Editorial: Inherited retinal dystrophies: a light at the end of the tunnel? Frontiers in Cell and Developmental Biology. 11: 1301279
Grabinski SE, Parsana D, Perkins BD. (2023) Comparative analysis of transcriptional changes in zebrafish and mutants by RNA-seq reveals upregulation of inflammatory and stress-related pathways. Frontiers in Molecular Neuroscience. 16: 1148840
Wang J, Thomas HR, Thompson RG, et al. (2022) Variable phenotypes and penetrance between and within different zebrafish ciliary transition zone mutants. Disease Models & Mechanisms. 15
Perkins BD. (2022) Zebrafish models of inherited retinal dystrophies. Journal of Translational Genetics and Genomics. 6: 95-110
Fogerty J, Song P, Boyd P, et al. (2022) Notch inhibition promotes regeneration and immunosuppression supports cone survival in a zebrafish model of inherited retinal dystrophy. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Banerjee S, Ranspach LE, Luo X, et al. (2022) Vision and sensorimotor defects associated with loss of Vps11 function in a zebrafish model of genetic leukoencephalopathy. Scientific Reports. 12: 3511
Gulluni F, Prever L, Li H, et al. (2021) PI(3,4)P2-mediated cytokinetic abscission prevents early senescence and cataract formation. Science (New York, N.Y.). 374: eabk0410
Fogerty J, Perkins BD. (2021) Knockout of the CEP290 gene in human induced pluripotent stem cells. Stem Cell Research. 52: 102243
Song P, Fogerty J, Cianciolo LT, et al. (2020) Cone Photoreceptor Degeneration and Neuroinflammation in the Zebrafish Bardet-Biedl Syndrome 2 () Mutant Does Not Lead to Retinal Regeneration. Frontiers in Cell and Developmental Biology. 8: 578528
Pollock LM, Perkins B, Anand-Apte B. (2020) Primary cilia are present on endothelial cells of the hyaloid vasculature but are not required for the development of the blood-retinal barrier. Plos One. 15: e0225351
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