Nail Burnashev

Affiliations: 
Vrije Universiteit Amsterdam, Amsterdam, Netherlands 
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"Nail Burnashev"
Mean distance: 13.25 (cluster 6)
 
SNBCP

Children

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Rogier Min grad student 2003-2007 VU Amsterdam
Marat Mukhamedyarov research scientist 2014-2014
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Publications

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Bertocchi I, Eltokhi A, Rozov A, et al. (2021) Voltage-independent GluN2A-type NMDA receptor Ca signaling promotes audiogenic seizures, attentional and cognitive deficits in mice. Communications Biology. 4: 59
Salmi M, Del Gallo F, Minlebaev M, et al. (2019) Impaired vocal communication, sleep-related discharges, and transient alteration of slow-wave sleep in developing mice lacking the GluN2A subunit of N-methyl-d-aspartate receptors. Epilepsia
Fernandez A, Dumon C, Guimond D, et al. (2018) The GABA Developmental Shift Is Abolished by Maternal Immune Activation Already at Birth. Cerebral Cortex (New York, N.Y. : 1991)
Pons-Bennaceur A, Tsintsadze V, Bui TT, et al. (2018) Diadenosine-Polyphosphate Analogue AppCH2ppA Suppresses Seizures by Enhancing Adenosine Signaling in the Cortex. Cerebral Cortex (New York, N.Y. : 1991)
Salmi M, Bolbos R, Bauer S, et al. (2018) Transient microstructural brain anomalies and epileptiform discharges in mice defective for epilepsy and language-related NMDA receptor subunit gene Grin2a. Epilepsia
Bannerman DM, Borchardt T, Jensen V, et al. (2018) Somatic Accumulation of GluA1-AMPA Receptors Leads to Selective Cognitive Impairments in Mice. Frontiers in Molecular Neuroscience. 11: 199
Sibarov DA, Bruneau N, Antonov SM, et al. (2017) Functional Properties of Human NMDA Receptors Associated with Epilepsy-Related Mutations of GluN2A Subunit. Frontiers in Cellular Neuroscience. 11: 155
Gataullina S, Lozovaya N, Burnashev N, et al. (2017) The role of altered NMDA receptors expression in the epilepsy related to Tuberos Sclerosis Complex European Journal of Paediatric Neurology. 21: e8
Viatchenko-Karpinski V, Novosolova N, Ishchenko Y, et al. (2016) Stable, synthetic analogs of diadenosine tetraphosphate inhibit rat and human P2X3 receptors and inflammatory pain. Molecular Pain. 12
Valiullina F, Zakharova Y, Mukhtarov M, et al. (2016) The Relative Contribution of NMDARs to Excitatory Postsynaptic Currents is Controlled by Ca(2+)-Induced Inactivation. Frontiers in Cellular Neuroscience. 10: 12
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