Susan Waltz
Affiliations: | Cancer Biology | University of Cincinnati, Cincinnati, OH |
Area:
Cell BiologyGoogle:
"Susan Waltz"Parents
Sign in to add mentorMichael Leffak | grad student | 1989-1994 | Biomedical Sciences |
Sandra J. Degen | post-doc | 1994-1997 | Cincinnati Children's Hospital Medical Center (Chemistry Tree) |
Children
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Publications
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Vicente-Muñoz S, Davis JC, Lane A, et al. (2024) Lipid profiling of RON and DEK-dependent signaling in breast cancer guides discovery of gene networks predictive of poor outcomes. Frontiers in Oncology. 14: 1382986 |
Davis JC, Waltz SE. (2024) The MET Family of Receptor Tyrosine Kinases Promotes a Shift to Pro-Tumor Metabolism. Genes. 15 |
Oropeza E, Seker S, Carrel S, et al. (2023) Molecular portraits of cell cycle checkpoint kinases in cancer evolution, progression, and treatment responsiveness. Science Advances. 9: eadf2860 |
Hunt BG, Davis JC, Fox LH, et al. (2023) RON-augmented cholesterol biosynthesis in breast cancer metastatic progression and recurrence. Oncogene |
Hunt BG, Fox LH, Davis JC, et al. (2023) An Introduction and Overview of RON Receptor Tyrosine Kinase Signaling. Genes. 14 |
Vicente-Muñoz S, Hunt BG, Lange TE, et al. (2022) NMR-based metabolomic analysis identifies RON-DEK-β-catenin dependent metabolic pathways and a gene signature that stratifies breast cancer patient survival. Plos One. 17: e0274128 |
Brown NE, Jones A, Hunt BG, et al. (2022) Prostate tumor RON receptor signaling mediates macrophage recruitment to drive androgen deprivation therapy resistance through Gas6-mediated Axl and RON signaling. The Prostate |
Hunt BG, Jones A, Lester C, et al. (2022) RON () and HGFL () Co-Overexpression Supports Breast Tumorigenesis through Autocrine and Paracrine Cellular Crosstalk. Cancers. 14 |
Ruiz-Torres SJ, Bourn JR, Benight NM, et al. (2021) Macrophage-mediated RON signaling supports breast cancer growth and progression through modulation of IL-35. Oncogene |
Bourn JR, Ruiz-Torres SJ, Hunt BG, et al. (2021) Tumor cell intrinsic RON signaling suppresses innate immune responses in breast cancer through inhibition of IRAK4 signaling. Cancer Letters. 503: 75-90 |