Ricardo Tapia

Instituto de Fisiologia Celular UNAM, Ciudad de México, Ciudad de México, Mexico 
Epilepsy, neurodegenerative diseases, and neuronal death
"Ricardo Tapia"
Mean distance: 17.09 (cluster 46)
Cross-listing: Neuropathology Tree

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Netzahualcoyotzi C, Tapia R. (2018) Tetanus toxin C-fragment protects against excitotoxic spinal motoneuron degeneration in vivo. Scientific Reports. 8: 16584
Ramírez-Jarquín UN, Tapia R. (2018) Excitatory and inhibitory neuronal circuits in the spinal cord and their role in the control of motor neuron function and degeneration. Acs Chemical Neuroscience
Lazo-Gomez R, Tapia R. (2017) Quercetin prevents spinal motor neuron degeneration induced by chronic excitotoxic stimulus by a sirtuin 1-dependent mechanism. Translational Neurodegeneration. 6: 31
Ramírez-Jarquín UN, Tapia R. (2017) Chronic GABAergic blockade in the spinal cord in vivo induces motor alterations and neurodegeneration. Neuropharmacology
Ramírez-Jarquín UN, Rojas F, van Zundert B, et al. (2017) Chronic Infusion of SOD1(G93A) Astrocyte-Secreted Factors Induces Spinal Motoneuron Degeneration and Neuromuscular Dysfunction in Healthy Rats. Journal of Cellular Physiology
Ramírez-Jarquín UN, Tapia R. (2016) Neuropathological characterization of spinal motor neuron degeneration processes induced by acute and chronic excitotoxic stimulus in vivo. Neuroscience
Lazo-Gómez R, Tapia R. (2016) Motor Alterations Induced by Chronic 4-Aminopyridine Infusion in the Spinal Cord In vivo: Role of Glutamate and GABA Receptors. Frontiers in Neuroscience. 10: 200
Santa-Cruz LD, Guerrero-Castillo S, Uribe-Carvajal S, et al. (2016) Mitochondrial dysfunction during the early stages of excitotoxic spinal motor neuron degeneration in vivo. Acs Chemical Neuroscience
Salazar P, Tapia R. (2015) Epilepsy and hippocampal neurodegeneration induced by glutamate decarboxylase inhibitors in awake rats. Epilepsy Research. 116: 27-33
Netzahualcoyotzi C, Tapia R. (2015) Degeneration of spinal motor neurons by chronic AMPA-induced excitotoxicity in vivo and protection by energy substrates. Acta Neuropathologica Communications. 3: 27
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