Glenn J. Giesler

Affiliations: 
University of Minnesota, Twin Cities, Minneapolis, MN 
Area:
Pain
Google:
"Glenn Giesler"
Mean distance: 14.07 (cluster 6)
 
SNBCP
BETA: Related publications

Publications

You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Khasabov SG, Truong H, Rogness VM, et al. (2020) Responses of neurons in the primary somatosensory cortex to itch- and pain-producing stimuli in rats. Journal of Neurophysiology
Lipshetz B, Khasabov SG, Truong H, et al. (2018) Responses of thalamic neurons to itch- and pain-producing stimuli in rats. Journal of Neurophysiology
Lipshetz B, Giesler GJ. (2015) Effects of scratching and other counter-stimuli on responses of trigeminothalamic tract neurons to itch-inducing stimuli in rats. Journal of Neurophysiology. jn.00326.2015
Jansen NA, Giesler GJ. (2015) Response characteristics of pruriceptive and nociceptive trigeminoparabrachial tract neurons in the rat. Journal of Neurophysiology. 113: 58-70
Moser HR, Giesler GJ. (2014) Itch elicited by intradermal injection of serotonin, intracisternal injection of morphine, and their synergistic interactions in rats. Neuroscience. 274: 119-27
Moser HR, Giesler GJ. (2014) Characterization of pruriceptive trigeminothalamic tract neurons in rats. Journal of Neurophysiology. 111: 1574-89
Moser HR, Giesler GJ. (2013) Itch and analgesia resulting from intrathecal application of morphine: contrasting effects on different populations of trigeminothalamic tract neurons. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 33: 6093-101
Davidson S, Zhang X, Khasabov SG, et al. (2012) Pruriceptive spinothalamic tract neurons: physiological properties and projection targets in the primate. Journal of Neurophysiology. 108: 1711-23
Davidson S, Giesler GJ. (2010) The multiple pathways for itch and their interactions with pain. Trends in Neurosciences. 33: 550-8
Duvick L, Barnes J, Ebner B, et al. (2010) SCA1-like disease in mice expressing wild-type ataxin-1 with a serine to aspartic acid replacement at residue 776. Neuron. 67: 929-35
See more...