Olav Olsen

Genentech, Inc., San Francisco, CA, United States 
"Olav Olsen"
Mean distance: 14.03 (cluster 11)


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David Bredt post-doc 2001-2007 UCSF
Marc Tessier-Lavigne research scientist 2008- Genentech, Inc.
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Kwart D, Gregg A, Scheckel C, et al. (2019) A Large Panel of Isogenic APP and PSEN1 Mutant Human iPSC Neurons Reveals Shared Endosomal Abnormalities Mediated by APP β-CTFs, Not Aβ. Neuron
Hertz NT, Adams EL, Weber RA, et al. (2019) Neuronally Enriched RUFY3 Is Required for Caspase-Mediated Axon Degeneration. Neuron
Wu Z, Makihara S, Yam PT, et al. (2019) Long-Range Guidance of Spinal Commissural Axons by Netrin1 and Sonic Hedgehog from Midline Floor Plate Cells. Neuron
Marik SA, Olsen O, Tessier-Lavigne M, et al. (2016) Physiological role for amyloid precursor protein in adult experience-dependent plasticity. Proceedings of the National Academy of Sciences of the United States of America
Renier N, Adams EL, Kirst C, et al. (2016) Mapping of Brain Activity by Automated Volume Analysis of Immediate Early Genes. Cell
Simon DJ, Pitts J, Hertz NT, et al. (2016) Axon Degeneration Gated by Retrograde Activation of Somatic Pro-apoptotic Signaling. Cell. 164: 1031-45
Hernandez-Enriquez B, Wu Z, Martinez E, et al. (2015) Floor plate-derived neuropilin-2 functions as a secreted semaphorin sink to facilitate commissural axon midline crossing. Genes & Development. 29: 2617-32
Xu K, Olsen O, Tzvetkova-Robev D, et al. (2015) The crystal structure of DR6 in complex with the amyloid precursor protein provides insight into death receptor activation. Genes & Development. 29: 785-90
Hernandez-Enriquez B, Wu Z, Martinez E, et al. (2015) Floor plate-derived neuropilin-2 functions as a secreted semaphoring sink to facilitate commissural axon midline crossing Genes and Development. 29: 2617-2632
Olsen O, Kallop DY, McLaughlin T, et al. (2014) Genetic analysis reveals that amyloid precursor protein and death receptor 6 function in the same pathway to control axonal pruning independent of β-secretase. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 6438-47
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