David B. Jaffe

Affiliations: 
University of Texas, San Antonio, San Antonio, TX, United States 
Area:
Hippocampus LTP
Website:
http://bio.utsa.edu/labpages/jaffe/people.html
Google:
"David Jaffe"
Mean distance: 14.11 (cluster 6)
 
SNBCP
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Publications

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Wang B, Bugay V, Ling L, et al. (2016) Knockout of the BK β4 subunit promotes a functional coupling of BK channels and ryanodine receptors that mediate a fAHP-induced increase in excitability. Journal of Neurophysiology. jn.00857.2015
Wang B, Jaffe DB, Brenner R. (2014) Current understanding of iberiotoxin-resistant BK channels in the nervous system. Frontiers in Physiology. 5: 382
Du X, Hao H, Gigout S, et al. (2014) Control of somatic membrane potential in nociceptive neurons and its implications for peripheral nociceptive transmission. Pain. 155: 2306-22
Jaffe DB, Wang B, Brenner R. (2011) Shaping of action potentials by type I and type II large-conductance Ca²+-activated K+ channels. Neuroscience. 192: 205-18
Hemond P, Migliore M, Ascoli GA, et al. (2009) The membrane response of hippocampal CA3b pyramidal neurons near rest: Heterogeneity of passive properties and the contribution of hyperpolarization-activated currents. Neuroscience. 160: 359-70
Hemond P, Epstein D, Boley A, et al. (2008) Distinct classes of pyramidal cells exhibit mutually exclusive firing patterns in hippocampal area CA3b. Hippocampus. 18: 411-24
Jaffe DB, Gutiérrez R. (2007) Mossy fiber synaptic transmission: communication from the dentate gyrus to area CA3. Progress in Brain Research. 163: 109-32
Zaika O, Tolstykh GP, Jaffe DB, et al. (2007) Inositol triphosphate-mediated Ca2+ signals direct purinergic P2Y receptor regulation of neuronal ion channels. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 27: 8914-26
Gamper N, Zaika O, Li Y, et al. (2006) Oxidative modification of M-type K(+) channels as a mechanism of cytoprotective neuronal silencing. The Embo Journal. 25: 4996-5004
Zaika O, Lara LS, Gamper N, et al. (2006) Angiotensin II regulates neuronal excitability via phosphatidylinositol 4,5-bisphosphate-dependent modulation of Kv7 (M-type) K+ channels. The Journal of Physiology. 575: 49-67
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