Jason W. Mastaitis

Affiliations: 
Internal Medicine/Endocrinology Yale University, New Haven, CT 
Area:
Diabetes, Hypoglycemia
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"Jason Mastaitis"
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Publications

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Altarejos JY, Pangilinan J, Podgrabinska S, et al. (2023) Preclinical, randomized phase 1, and compassionate use evaluation of REGN4461, a leptin receptor agonist antibody for leptin deficiency. Science Translational Medicine. 15: eadd4897
Adam RC, Pryce DS, Lee JS, et al. (2023) Activin E-ACVR1C cross talk controls energy storage via suppression of adipose lipolysis in mice. Proceedings of the National Academy of Sciences of the United States of America. 120: e2309967120
Mastaitis J, Eckersdorff M, Min S, et al. (2015) Loss of SFRP4 Alters Body Size, Food Intake and Energy Expenditure in Diet-Induced Obese Male Mice. Endocrinology. en20151257
Fan X, Chan O, Ding Y, et al. (2015) Reduction in SGLT1 mRNA Expression in the Ventromedial Hypothalamus Improves the Counterregulatory Responses to Hypoglycemia in Recurrently Hypoglycemic and Diabetic Rats. Diabetes
Mastaitis J, Min S, Elvert R, et al. (2015) GPR17 gene disruption does not alter food intake or glucose homeostasis in mice. Proceedings of the National Academy of Sciences of the United States of America. 112: 1845-9
Mobbs CV, Mastaitis J, Isoda F, et al. (2013) Treatment of diabetes and diabetic complications with a ketogenic diet. Journal of Child Neurology. 28: 1009-14
Yang L, Isoda F, Yen K, et al. (2012) Hypothalamic Fkbp51 is induced by fasting, and elevated hypothalamic expression promotes obese phenotypes. American Journal of Physiology. Endocrinology and Metabolism. 302: E987-91
Poplawski MM, Mastaitis JW, Isoda F, et al. (2011) Reversal of diabetic nephropathy by a ketogenic diet. Plos One. 6: e18604
Poplawski MM, Mastaitis JW, Mobbs CV. (2011) Naloxone, but not valsartan, preserves responses to hypoglycemia after antecedent hypoglycemia: role of metabolic reprogramming in counterregulatory failure. Diabetes. 60: 39-46
Poplawski MM, Mastaitis JW, Yang XJ, et al. (2010) Hypothalamic responses to fasting indicate metabolic reprogramming away from glycolysis toward lipid oxidation. Endocrinology. 151: 5206-17
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