Michael F. Waters
Affiliations: | 1999 | Biochemistry and Molecular Biology | University of Florida, Gainesville, Gainesville, FL, United States |
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| Khare S, Galeano K, Zhang Y, et al. (2018) C-terminal proline deletions in KCNC3 cause delayed channel inactivation and an adult-onset progressive SCA13 with spasticity. Cerebellum (London, England) |
| Khare S, Nick JA, Zhang Y, et al. (2017) A KCNC3 mutation causes a neurodevelopmental, non-progressive SCA13 subtype associated with dominant negative effects and aberrant EGFR trafficking. Plos One. 12: e0173565 |
| Bickford JS, Ali NF, Nick JA, et al. (2014) Endothelin-1-mediated vasoconstriction alters cerebral gene expression in iron homeostasis and eicosanoid metabolism. Brain Research. 1588: 25-36 |
| Gallego-Iradi C, Bickford JS, Khare S, et al. (2014) KCNC3(R420H), a K(+) channel mutation causative in spinocerebellar ataxia 13 displays aberrant intracellular trafficking. Neurobiology of Disease. 71: 270-9 |
| Middlebrooks JC, Nick HS, Subramony SH, et al. (2013) Mutation in the kv3.3 voltage-gated potassium channel causing spinocerebellar ataxia 13 disrupts sound-localization mechanisms. Plos One. 8: e76749 |
| Figueroa KP, Minassian NA, Stevanin G, et al. (2010) KCNC3: phenotype, mutations, channel biophysics-a study of 260 familial ataxia patients. Human Mutation. 31: 191-6 |
| Mellott JK, Nick HS, Waters MF, et al. (2001) Cytokine-induced changes in chromatin structure and in vivo footprints in the inducible NOS promoter. American Journal of Physiology. Lung Cellular and Molecular Physiology. 280: L390-9 |