Noel R. Rose
Affiliations: | Johns Hopkins University, Baltimore, MD |
Area:
Immunology, Microbiology BiologyGoogle:
"Noel Rose"Children
Sign in to add trainee| Marina G. Afanasyeva | grad student | 2002 | Johns Hopkins |
| Shey-Cherng Tzou | grad student | 2008 | Johns Hopkins |
| Davinna L. Ligons | grad student | 2009 | Johns Hopkins |
| Geral C. Baldeviano | grad student | 2010 | Johns Hopkins |
| Jobert G. Barin | grad student | 2010 | Johns Hopkins |
| Melissa A. Landek-Salgado | grad student | 2010 | Johns Hopkins |
| Leora J. Vegosen | grad student | 2012 | Johns Hopkins |
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Publications
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| Langan D, Rose NR, Moudgil KD. (2020) Common innate pathways to autoimmune disease. Clinical Immunology (Orlando, Fla.). 212: 108361 |
| Cottagiri M, Nyandjo M, Stephens M, et al. (2018) In drug-induced, immune-mediated hepatitis, interleukin-33 reduces hepatitis and improves survival independently and as a consequence of FoxP3+ T-cell activity. Cellular & Molecular Immunology |
| Rose NR. (2017) Negative selection, epitope mimicry and autoimmunity. Current Opinion in Immunology. 49: 51-55 |
| Barin JG, Talor MV, Diny NL, et al. (2017) Regulation of autoimmune myocarditis by host responses to the microbiome. Experimental and Molecular Pathology |
| Tsokos GC, Rose NR. (2017) Immune cell signaling in autoimmune diseases. Clinical Immunology (Orlando, Fla.). 181: 1-8 |
| Diny NL, Rose NR, Čiháková D. (2017) Eosinophils in Autoimmune Diseases. Frontiers in Immunology. 8: 484 |
| Fontes JA, Barin JG, Talor MV, et al. (2017) Complete Freund's adjuvant induces experimental autoimmune myocarditis by enhancing IL-6 production during initiation of the immune response. Immunity, Inflammation and Disease. 5: 163-176 |
| Diny NL, Baldeviano GC, Talor MV, et al. (2017) Eosinophil-derived IL-4 drives progression of myocarditis to inflammatory dilated cardiomyopathy. The Journal of Experimental Medicine |
| Ong S, Rose NR, Cihakova D. (2016) Natural killer cells in inflammatory heart disease. Clinical Immunology (Orlando, Fla.) |
| Diny NL, Hou X, Barin JG, et al. (2016) Macrophages and cardiac fibroblasts are the main producers of eotaxins and regulate eosinophil trafficking to the heart. European Journal of Immunology |