Gerhard Rammes

Affiliations: 
Pharmacology Technische Universität München (TUM), München, Bayern, Germany 
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"Gerhard Rammes"
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Hofmann C, Sander A, Wang XX, et al. (2021) Inhalational Anesthetics Do Not Deteriorate Amyloid-β-Derived Pathophysiology in Alzheimer's Disease: Investigations on the Molecular, Neuronal, and Behavioral Level. Journal of Alzheimer's Disease : Jad. 84: 1193-1218
Rammes G, Parsons CG. (2020) The Aβ aggregation modulator MRZ-99030 prevents and even reverses synaptotoxic effects of Aβ on LTP even following serial dilution to a 500:1 stoichiometric excess of Aβ, suggesting a beneficial prion-like seeding mechanism. Neuropharmacology. 108267
Pigoni M, Hsia HE, Hartmann J, et al. (2020) Seizure protein 6 controls glycosylation and trafficking of kainate receptor subunits GluK2 and GluK3. The Embo Journal. e103457
Bürge M, Kratzer S, Mattusch C, et al. (2019) The anaesthetic xenon partially restores an amyloid beta-induced impairment in murine hippocampal synaptic plasticity. Neuropharmacology
Ryskamp D, Wu L, Wu J, et al. (2018) Pridopidine stabilizes mushroom spines in mouse models of Alzheimer's disease by acting on the sigma-1 receptor. Neurobiology of Disease
Schmid S, Rammes G, Blobner M, et al. (2018) Cognitive decline in Tg2576 mice shows sex-specific differences and correlates with cerebral amyloid-beta. Behavioural Brain Research
Rammes G, Seeser F, Mattusch K, et al. (2018) The NMDA receptor antagonist Radiprodil reverses the synaptotoxic effects of different amyloid-beta (Aβ) species on long-term potentiation (LTP). Neuropharmacology
Kratzer S, Mattusch C, Garcia PS, et al. (2017) Propofol and Sevoflurane Differentially Modulate Cortical Depolarization following Electric Stimulation of the Ventrobasal Thalamus. Frontiers in Computational Neuroscience. 11: 109
Irl H, Kratzer S, Schwerin S, et al. (2017) Tranexamic acid impairs hippocampal synaptic transmission mediated by gamma aminobutyric acid receptor type A. European Journal of Pharmacology
Dedic N, Pöhlmann ML, Richter JS, et al. (2017) Cross-disorder risk gene CACNA1C differentially modulates susceptibility to psychiatric disorders during development and adulthood. Molecular Psychiatry
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