Charles K. Abrams
Affiliations: | Albert Einstein College of Medicine, New York, New York, United States |
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"Charles Abrams"Mean distance: 15.21 (cluster 32) | S | N | B | C | P |
Children
Sign in to add trainee| Ian Stuart Kellar | grad student | 2000-2004 | University of Sussex (United Kingdom) (PsychTree) |
| Rafael E Flores-Obando | grad student | 2011-2017 | SUNY Downstate Medical Center |
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Publications
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| Abrams CK. (2023) Mechanisms of Diseases Associated with Mutation in GJC2/Connexin 47. Biomolecules. 13 |
| Flores-Obando RE, Freidin MM, Hernández AI, et al. (2022) Activation of the unfolded protein response by Connexin47 mutations associated with Pelizaeus-Merzbacher-like disease. Molecular and Cellular Neurosciences. 103716 |
| Abrams CK, Flores-Obando RE, Dungan GD, et al. (2021) Investigating oligodendrocyte connexins: Heteromeric interactions between Cx32 and mutant or wild-type forms of Cx47 do not contribute to or modulate gap junction function. Glia |
| Papaneophytou CP, Georgiou E, Karaiskos C, et al. (2018) Regulatory role of oligodendrocyte gap junctions in inflammatory demyelination. Glia |
| Alaei SR, Abrams CK, Bulinski JC, et al. (2018) Acetylation of C-terminal lysines modulates protein turnover and stability of Connexin-32. Bmc Cell Biology. 19: 22 |
| Abrams CK, Peinado A, Mahmoud R, et al. (2018) Alterations at Arg76 of human connexin 46, a residue associated with cataract formation, cause loss of gap junction formation but preserve hemichannel function. American Journal of Physiology. Cell Physiology |
| Flores-Obando RE, Freidin MM, Abrams CK. (2018) Rapid and Specific Immunomagnetic Isolation of Mouse Primary Oligodendrocytes. Journal of Visualized Experiments : Jove |
| Abrams CK. (2017) Diseases of connexins expressed in myelinating glia. Neuroscience Letters |
| Abrams CK, Goman M, Wong S, et al. (2017) Loss of Coupling Distinguishes GJB1 Mutations Associated with CNS Manifestations of CMT1X from Those Without CNS Manifestations. Scientific Reports. 7: 40166 |
| Olympiou M, Sargiannidou I, Markoullis K, et al. (2016) Systemic inflammation disrupts oligodendrocyte gap junctions and induces ER stress in a model of CNS manifestations of X-linked Charcot-Marie-Tooth disease. Acta Neuropathologica Communications. 4: 95 |