Angelika Doetzlhofer

Affiliations: 
Neuroscience Johns Hopkins University, Baltimore, MD 
Area:
Inner Ear Development
Website:
http://neuroscience.jhu.edu/AngelikaDoetzlhofer.php
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"Angelika Doetzlhofer"
Mean distance: 16.09 (cluster 11)
 
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Publications

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Evsen L, Li X, Zhang S, et al. (2020) miRNAs inhibit CHD7 expression and control auditory-sensory progenitor cell behavior in the developing inner ear. Development (Cambridge, England). 147
Basch ML, Brown RM, Jen HI, et al. (2016) Fine-tuning of Notch signaling sets the boundary of the organ of Corti and establishes sensory cell fates. Elife. 5
Evsen L, Doetzlhofer A. (2016) Gene Transfer into the Chicken Auditory Organ by In Ovo Micro-electroporation. Journal of Visualized Experiments : Jove
Campbell DP, Chrysostomou E, Doetzlhofer A. (2016) Canonical Notch signaling plays an instructive role in auditory supporting cell development. Scientific Reports. 6: 19484
Basch ML, Brown RM, Jen H, et al. (2016) Author response: Fine-tuning of Notch signaling sets the boundary of the organ of Corti and establishes sensory cell fates Elife
Golden EJ, Benito-Gonzalez A, Doetzlhofer A. (2015) The RNA-binding protein LIN28B regulates developmental timing in the mammalian cochlea Proceedings of the National Academy of Sciences of the United States of America. 112: E3864-E3873
Korrapati S, Roux I, Glowatzki E, et al. (2013) Notch signaling limits supporting cell plasticity in the hair cell-damaged early postnatal murine cochlea. Plos One. 8: e73276
Doetzlhofer A, Basch ML, Ohyama T, et al. (2009) Hey2 regulation by FGF provides a Notch-independent mechanism for maintaining pillar cell fate in the organ of Corti. Developmental Cell. 16: 58-69
Laine H, Doetzlhofer A, Mantela J, et al. (2007) p19(Ink4d) and p21(Cip1) collaborate to maintain the postmitotic state of auditory hair cells, their codeletion leading to DNA damage and p53-mediated apoptosis. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 27: 1434-44
White PM, Doetzlhofer A, Lee YS, et al. (2006) Mammalian cochlear supporting cells can divide and trans-differentiate into hair cells. Nature. 441: 984-7
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