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Matthew P. Frosch, MD PhD

Affiliations: 
Pathology Massachusetts General Hospital and Harvard University, Boston, MA, United States 
Area:
cerebral amyloid angiopathy
Website:
http://hst.mit.edu/public/people/faculty/facultyBiosketch.jsp?key=Frosch
Google:
"Matthew Frosch"
Bio:

http://madrc.mgh.harvard.edu/matthew-p-frosch-md-phd



Mean distance: 16.76 (cluster 24)
 
Cross-listing: Neuropathology Tree - Alzheimer's Tree

BETA: Related publications

Publications

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Schlunk F, Böhm M, Boulouis G, et al. (2019) Secondary Bleeding During Acute Experimental Intracerebral Hemorrhage. Stroke. 50: 1210-1215
Kunkle BW, Grenier-Boley B, Sims R, et al. (2019) Genetic meta-analysis of diagnosed Alzheimer's disease identifies new risk loci and implicates Aβ, tau, immunity and lipid processing. Nature Genetics. 51: 414-430
van Veluw SJ, Reijmer YD, van der Kouwe AJ, et al. (2019) Histopathology of diffusion imaging abnormalities in cerebral amyloid angiopathy. Neurology
Freeze WM, Bacskai BJ, Frosch MP, et al. (2019) Blood-Brain Barrier Leakage and Microvascular Lesions in Cerebral Amyloid Angiopathy. Stroke. STROKEAHA118023788
Pasi M, Charidimou A, Boulouis G, et al. (2019) Cerebral small vessel disease in patients with spontaneous cerebellar hemorrhage. Journal of Neurology
Dujardin S, Bégard S, Caillierez R, et al. (2018) Different tau species lead to heterogeneous tau pathology propagation and misfolding. Acta Neuropathologica Communications. 6: 132
Barroeta-Espar I, Weinstock LD, Perez-Nievas BG, et al. (2018) Distinct cytokine profiles in human brains resilient to Alzheimer's pathology. Neurobiology of Disease
Ly KI, Oakley DH, Pine AB, et al. (2018) Wide Range of Clinical Outcomes in Patients with Gliomatosis Cerebri Growth Pattern: A Clinical, Radiographic, and Histopathologic Study. The Oncologist
DeVos SL, Corjuc BT, Oakley DH, et al. (2018) Synaptic Tau Seeding Precedes Tau Pathology in Human Alzheimer's Disease Brain. Frontiers in Neuroscience. 12: 267
DeVos SL, Corjuc BT, Commins C, et al. (2018) Tau reduction in the presence of amyloid-β prevents tau pathology and neuronal death in vivo. Brain : a Journal of Neurology
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