Benvenuto Pernis
Affiliations: | Columbia University, New York, NY |
Area:
ImmunologyGoogle:
"Benvenuto Pernis"Bio:
(1923-2011)
https://www.legacy.com/us/obituaries/bostonglobe/name/benvenuto-pernis-obituary?id=21595010
DOI: 10.1002/eji.201270015
Mean distance: 15.11 (cluster 6)
Cross-listing: ImmunoTree - Chemistry Tree
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Publications
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Pernis B. (2005) Silica and the immune system. Acta Bio-Medica : Atenei Parmensis. 76: 38-44 |
Pernis B. (2001) [The immunologic etiology of silicosis. Part IIa: Biochemical and immunologic considerations on hyaline. 1958]. La Medicina Del Lavoro. 92: 461-7 |
Maruyama S, Cantu E, DeMartino C, et al. (1999) Interaction of baboon anti-alpha-galactosyl antibody with pig tissues. The American Journal of Pathology. 155: 1635-49 |
Maruyama S, Cantu E3rd, Pernis B, et al. (1999) Alpha-galactosyl antibody redistributes alpha-galactosyl at the surface of pig blood and endothelial cells. Transplant Immunology. 7: 101-6 |
Wang ZQ, Orlikowsky T, Dudhane A, et al. (1998) Staphylococcal enterotoxin B-induced T-cell anergy is mediated by regulatory T cells. Immunology. 94: 331-9 |
Jiang H, Kashleva H, Xu LX, et al. (1998) T cell vaccination induces T cell receptor Vbeta-specific Qa-1-restricted regulatory CD8(+) T cells. Proceedings of the National Academy of Sciences of the United States of America. 95: 4533-7 |
Jiang MH, Kashleva H, Yu B, et al. (1998) TCR Vβ specific, Qa-1 restricted CD8+ T hybridoma clones generated from T Cell Vaccinated (TCV) Faseb Journal. 12: A1092 |
Jiang H, Ware R, Stall A, et al. (1995) Murine CD8+ T cells that specifically delete autologous CD4+ T cells expressing V beta 8 TCR: a role of the Qa-1 molecule. Immunity. 2: 185-94 |
Ware R, Jiang H, Braunstein N, et al. (1995) Human CD8+ T lymphocyte clones specific for T cell receptor V beta families expressed on autologous CD4+ T cells. Immunity. 2: 177-84 |
Frumento G, de Totero D, Ferrara GB, et al. (1994) Cellular mechanisms of exogenous peptide binding to HLA class II molecules in B cells. Cellular Immunology. 155: 1-10 |