Anna Molander

University of Göteborg, Gothenburg, Västra Götalands län, Sweden 
Pharmacology of ethanol
"Anna Molander"
Mean distance: 19.18 (cluster 19)
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de la Cour C, Sørensen G, Wortwein G, et al. (2015) Enhanced self-administration of alcohol in muscarinic acetylcholine M4 receptor knockout mice. European Journal of Pharmacology. 746: 1-5
Karlsson RM, Adermark L, Molander A, et al. (2012) Reduced alcohol intake and reward associated with impaired endocannabinoid signaling in mice with a deletion of the glutamate transporter GLAST. Neuropharmacology. 63: 181-9
Molander A, Vengeliene V, Heilig M, et al. (2012) Brain-specific inactivation of the Crhr1 gene inhibits post-dependent and stress-induced alcohol intake, but does not affect relapse-like drinking. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology. 37: 1047-56
Vengeliene V, Bilbao A, Molander A, et al. (2008) Neuropharmacology of alcohol addiction. British Journal of Pharmacology. 154: 299-315
Molander A, Lidö HH, Löf E, et al. (2007) The glycine reuptake inhibitor Org 25935 decreases ethanol intake and preference in male wistar rats. Alcohol and Alcoholism (Oxford, Oxfordshire). 42: 11-8
Ericson M, Molander A, Stomberg R, et al. (2006) Taurine elevates dopamine levels in the rat nucleus accumbens; antagonism by strychnine. The European Journal of Neuroscience. 23: 3225-9
Molander A, Löf E, Stomberg R, et al. (2005) Involvement of accumbal glycine receptors in the regulation of voluntary ethanol intake in the rat. Alcoholism, Clinical and Experimental Research. 29: 38-45
Molander A, Söderpalm B. (2005) Accumbal strychnine-sensitive glycine receptors: an access point for ethanol to the brain reward system. Alcoholism, Clinical and Experimental Research. 29: 27-37
Molander A, Söderpalm B. (2005) Glycine receptors regulate dopamine release in the rat nucleus accumbens. Alcoholism, Clinical and Experimental Research. 29: 17-26
Ericson M, Molander A, Löf E, et al. (2003) Ethanol elevates accumbal dopamine levels via indirect activation of ventral tegmental nicotinic acetylcholine receptors. European Journal of Pharmacology. 467: 85-93
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