Ruth Empson, PhD

Affiliations: 
University of Otago, Dunedin, Otago, New Zealand 
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"Ruth Empson"
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Publications

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Empson RM, Huang H, Nagaraja RY, et al. (2013) Enhanced synaptic inhibition in the cerebellar cortex of the ataxic PMCA2(-/-) knockout mouse. Cerebellum (London, England). 12: 667-75
Empson RM, Knöpfel T. (2012) Functional integration of calcium regulatory mechanisms at Purkinje neuron synapses. Cerebellum (London, England). 11: 640-50
Empson RM, Turner PR, Nagaraja RY, et al. (2010) Reduced expression of the Ca(2+) transporter protein PMCA2 slows Ca(2+) dynamics in mouse cerebellar Purkinje neurones and alters the precision of motor coordination. The Journal of Physiology. 588: 907-22
Jensen TP, Buckby LE, Empson RM. (2009) Reduced expression of the "fast" calcium transporter PMCA2a during homeostatic plasticity. Molecular and Cellular Neurosciences. 41: 364-72
Empson RM, Garside ML, Knöpfel T. (2007) Plasma membrane Ca2+ ATPase 2 contributes to short-term synapse plasticity at the parallel fiber to Purkinje neuron synapse. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 27: 3753-8
Jensen TP, Filoteo AG, Knopfel T, et al. (2007) Presynaptic plasma membrane Ca2+ ATPase isoform 2a regulates excitatory synaptic transmission in rat hippocampal CA3. The Journal of Physiology. 579: 85-99
Jensen TP, Buckby LE, Empson RM. (2004) Expression of plasma membrane Ca2+ ATPase family members and associated synaptic proteins in acute and cultured organotypic hippocampal slices from rat. Brain Research. Developmental Brain Research. 152: 129-36
Leckie C, Empson R, Becchetti A, et al. (2003) The NO pathway acts late during the fertilization response in sea urchin eggs. The Journal of Biological Chemistry. 278: 12247-54
Empson RM, Jefferys JG. (2001) Ca(2+) entry through L-type Ca(2+) channels helps terminate epileptiform activity by activation of a Ca(2+) dependent afterhyperpolarisation in hippocampal CA3. Neuroscience. 102: 297-306
Barrow PA, Empson RM, Gladwell SJ, et al. (2000) Functional phenotype in transgenic mice expressing mutant human presenilin-1. Neurobiology of Disease. 7: 119-26
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