Christopher Rhodes
Affiliations: | Neurobiology | University of Chicago, Chicago, IL |
Area:
Neuroscience Biology, Endocrinology Biology, Physiology BiologyGoogle:
"Christopher Rhodes"Mean distance: 26716.5
Children
Sign in to add trainee| Wenshuo Zhang | grad student | 2011 | Chicago |
| Alyssa K. Arata | grad student | 2012 | Chicago |
| Wilfredo Rosario | grad student | 2014 | Chicago |
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Publications
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| Yau B, Hays L, Liang C, et al. (2020) A fluorescent timer reporter enables sorting of insulin secretory granules by age. The Journal of Biological Chemistry |
| Waise TMZ, Rasti M, Duca FA, et al. (2019) Inhibition of upper small intestinal mTOR lowers plasma glucose levels by inhibiting glucose production. Nature Communications. 10: 714 |
| Hemnes AR, Luther JM, Rhodes CJ, et al. (2019) Human PAH is characterized by a pattern of lipid-related insulin resistance. Jci Insight. 4 |
| Boland BB, Brown C, Boland ML, et al. (2018) Pancreatic β-Cell Rest Replenishes Insulin Secretory Capacity and Attenuates Diabetes in an Extreme Model of Obese Type 2 Diabetes. Diabetes |
| Boland BB, Brown C, Alarcon C, et al. (2017) β-cell Control of Insulin Production During Starvation-Refeeding in Male Rats. Endocrinology |
| Wasserfall C, Nick HS, Campbell-Thompson M, et al. (2017) Persistence of Pancreatic Insulin mRNA Expression and Proinsulin Protein in Type 1 Diabetes Pancreata. Cell Metabolism. 26: 568-575.e3 |
| Rosario W, Singh I, Wautlet A, et al. (2016) The Brain to Pancreatic Islet Neuronal Map Reveals Differential Glucose Regulation from Distinct Hypothalamic Regions. Diabetes |
| Boland BB, Alarcón C, Ali A, et al. (2015) Monomethylated-adenines potentiate glucose-induced insulin production and secretion via inhibition of phosphodiesterase activity in rat pancreatic islets. Islets. e1073435 |
| Alarcon C, Boland BB, Uchizono Y, et al. (2015) Pancreatic ß-Cell Adaptive Plasticity in Obesity Increases Insulin Production but Adversely Affects Secretory Function. Diabetes |
| Kebede MA, Oler AT, Gregg T, et al. (2014) SORCS1 is necessary for normal insulin secretory granule biogenesis in metabolically stressed β cells. The Journal of Clinical Investigation. 124: 4240-56 |