Peter Kind

Affiliations: 
University of Edinburgh, Edinburgh, Scotland, United Kingdom 
Area:
Developmental Neuroscience
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"Peter Kind"
Mean distance: 13.38 (cluster 6)
 
SNBCP

Parents

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Colin Blakemore grad student 1993 Oxford
Susan J. Hockfield post-doc 1993-1997 Yale
Colin Blakemore post-doc 1997-2000 Oxford

Children

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Tommas J. Ellender research assistant 2003-2004 Edinburgh
Chih-Yuan Chiang grad student 2011- Edinburgh
Sally Till post-doc 2006- Edinburgh
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Publications

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Booker SA, Domanski APF, Dando OR, et al. (2019) Altered dendritic spine function and integration in a mouse model of fragile X syndrome. Nature Communications. 10: 4813
Domanski APF, Booker SA, Wyllie DJA, et al. (2019) Cellular and synaptic phenotypes lead to disrupted information processing in Fmr1-KO mouse layer 4 barrel cortex. Nature Communications. 10: 4814
Asiminas A, Jackson AD, Louros SR, et al. (2019) Sustained correction of associative learning deficits after brief, early treatment in a rat model of Fragile X Syndrome. Science Translational Medicine. 11
Strehlow V, Heyne HO, Vlaskamp DRM, et al. (2018) GRIN2A-related disorders: genotype and functional consequence predict phenotype. Brain : a Journal of Neurology
McKay S, Ryan TJ, McQueen J, et al. (2018) The Developmental Shift of NMDA Receptor Composition Proceeds Independently of GluN2 Subunit-Specific GluN2 C-Terminal Sequences. Cell Reports. 25: 841-851.e4
Selvaraj BT, Livesey MR, Zhao C, et al. (2018) C9ORF72 repeat expansion causes vulnerability of motor neurons to Ca2+-permeable AMPA receptor-mediated excitotoxicity. Nature Communications. 9: 347
Booker SA, Loreth D, Gee AL, et al. (2018) Postsynaptic GABABRs Inhibit L-Type Calcium Channels and Abolish Long-Term Potentiation in Hippocampal Somatostatin Interneurons. Cell Reports. 22: 36-43
Thomson SR, Seo SS, Barnes SA, et al. (2017) Cell-Type-Specific Translation Profiling Reveals a Novel Strategy for Treating Fragile X Syndrome. Neuron. 95: 550-563.e5
Currie SP, Luz LL, Booker SA, et al. (2017) Reduced local input to fast-spiking interneurons in the somatosensory cortex in the GABAA γ2 R43Q mouse model of absence epilepsy. Epilepsia
Qiu J, McQueen J, Bilican B, et al. (2016) Evidence for evolutionary divergence of activity-dependent gene expression in developing neurons. Elife. 5
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