Alicia M. Pickrell, Ph.D.
Affiliations: | 2012 | Neuroscience (Medicine) | University of Miami, Coral Gables, FL |
Area:
Neuroscience Biology, Molecular Biology, AgingGoogle:
"Alicia Pickrell"Mean distance: 23.34
Parents
Sign in to add mentor| Carlos T. Moraes | grad student | 2012 | University of Miami | |
| (Mitochondrial involvement in neurodegeneration and aging.) | ||||
| Richard J Youle | post-doc | University of Miami (Chemistry Tree) | ||
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Publications
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| DeFoor N, Paul S, Li S, et al. (2023) Remdesivir increases mtDNA copy number causing mild alterations to oxidative phosphorylation. Scientific Reports. 13: 15339 |
| Guilhaume-Correa F, Pickrell AM, VandeVord PJ. (2023) The Imbalance of Astrocytic Mitochondrial Dynamics Following Blast-Induced Traumatic Brain Injury. Biomedicines. 11 |
| Sarraf SA, Shah HV, Kanfer G, et al. (2022) Loss of TAX1BP1-Directed Autophagy Results in Protein Aggregate Accumulation in the Brain. Molecular Cell. 82: 1383-1385 |
| Paul S, Pickrell AM. (2021) Hidden phenotypes of PINK1/Parkin knockout mice. Biochimica Et Biophysica Acta. General Subjects. 1865: 129871 |
| Sarraf SA, Shah HV, Kanfer G, et al. (2020) Loss of TAX1BP1-Directed Autophagy Results in Protein Aggregate Accumulation in the Brain. Molecular Cell |
| Fritsch LE, Moore ME, Sarraf SA, et al. (2019) Ubiquitin and Receptor Dependent Mitophagy Pathways and Their Implication in Neurodegeneration. Journal of Molecular Biology |
| Sarraf SA, Sideris DP, Giagtzoglou N, et al. (2019) PINK1/Parkin Influences Cell Cycle by Sequestering TBK1 at Damaged Mitochondria, Inhibiting Mitosis. Cell Reports. 29: 225-235.e5 |
| Carter FE, Moore ME, Pickrell AM. (2019) Methods to detect mitophagy in neurons during disease. Journal of Neuroscience Methods. 325: 108351 |
| Zhang Z, Pickrell A, Bleck C, et al. (2018) PO-125 Fis1 governs normal mitophagy in slow muscle during the low-intensity and long-period exercise Exercise Biochemistry Review. 1 |
| Pinto M, Pickrell AM, Wang X, et al. (2016) Transient mitochondrial DNA double strand breaks in mice cause accelerated aging phenotypes in a ROS-dependent but p53/p21-independent manner. Cell Death and Differentiation |